Showing 141 - 160 results of 42,770 for search '(((( ((b largest) OR (b large)) decrease ) OR ( i large decrease ))) OR ( i cases increased ))', query time: 1.48s Refine Results
  1. 141

    DataSheet_1_c-Rel Is the Pivotal NF-κB Subunit in Germinal Center Diffuse Large B-Cell Lymphoma: A LYSA Study.zip by Nathalie Faumont (7137200)

    Published 2021
    “…We found that strong c-Rel DNA-binding activity was mostly found in GCBs on two independent series of 48 DLBCLs and 66 DLBCLs, the latter issued from the GHEDI series. c-Rel DNA-binding activity was associated with increased REL mRNA expression. Extending the study to the whole GHEDI and Lenz DLBCL published series of 202 and 233 cases, it was found that the c-Rel gene expression profile (GEP) overlapped partially (12%) but only with the GCB GEP and not with the GEP of ABC-DLBCLs. …”
  2. 142

    Foldases and holdases each decrease the level of unfolded protein. by Adam MR de Graff (9767829)

    Published 2020
    “…The benefit of a large spare foldase capacity can be seen from the rapid rise of the unfolded level [<i>U</i>], up 4-fold for only a 2-fold increase in unfolding load (thin black lines). …”
  3. 143
  4. 144
  5. 145

    Deletion of murine <i>Rhoh</i> leads to de-repression of <i>Bcl-6</i> via decreased KAISO levels and accelerates a malignancy phenotype in a murine model of lymphoma by Hiroto Horiguchi (3215001)

    Published 2022
    “…RHOH was initially identified as a translocation partner with BCL-6 in non-Hodgkin lymphoma (NHL), and aberrant somatic hypermutation (SHM) in the 5ʹ untranslated region of the RHOH gene has also been detected in Diffuse Large B-Cell Lymphoma (DLBCL). Recent data suggest a correlation between RhoH expression and disease progression in Acute Myeloid Leukaemia (AML). …”
  6. 146
  7. 147

    DataSheet_1_Mutational Profile and Clonal Evolution of Relapsed/Refractory Diffuse Large B-Cell Lymphoma.xlsx by Boram Lee (549836)

    Published 2021
    “…The genes most frequently altered (> 20%) were (in decreasing order of frequency) CDKN2A, PIM1, CD79B, TP53, MYD88, MYC, BTG2, BTG1, CDKN2B, DTX1, CD58, ETV6, and IRF4. …”
  8. 148
  9. 149
  10. 150
  11. 151
  12. 152
  13. 153
  14. 154
  15. 155
  16. 156
  17. 157
  18. 158
  19. 159
  20. 160