Search alternatives:
ng decrease » nn decrease (Expand Search), we decrease (Expand Search), gy decreased (Expand Search)
mm decrease » nn decrease (Expand Search), mean decrease (Expand Search), gy decreased (Expand Search)
a decrease » _ decreased (Expand Search), _ decreases (Expand Search)
10 ng » 10 mg (Expand Search), 10 nm (Expand Search)
50 mm » 10 mm (Expand Search), 50 mg (Expand Search), 20 mm (Expand Search)
ng decrease » nn decrease (Expand Search), we decrease (Expand Search), gy decreased (Expand Search)
mm decrease » nn decrease (Expand Search), mean decrease (Expand Search), gy decreased (Expand Search)
a decrease » _ decreased (Expand Search), _ decreases (Expand Search)
10 ng » 10 mg (Expand Search), 10 nm (Expand Search)
50 mm » 10 mm (Expand Search), 50 mg (Expand Search), 20 mm (Expand Search)
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Predictive values of IC<sub>50</sub> in mM (FFAs) for vanadium compounds and <i>in vivo</i> observations IC<sub>50</sub> in mM (FFAs) and decreasing blood glucose level.
Published 2014“…<p>Predictive values of IC<sub>50</sub> in mM (FFAs) for vanadium compounds and <i>in vivo</i> observations IC<sub>50</sub> in mM (FFAs) and decreasing blood glucose level.…”
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eNOS phosphorylation increased intracellular ROS generation and decreased cell viability in BH4-depleted cells.
Published 2015“…HEK293 eNOS control, *, <i>p</i> < 0.01). 50ng/mL VEGF caused further decrease of cell viability in eNOS overexpressing HEK293 cells treated with 10mM DAHP (VEGF plus DAHP vs. …”
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Olaparib reverses the oxLDL-mediated decrease in co-immunoprecipitation of IκBα with RelA or p50.
Published 2024“…Relative band densities from three independent experiments were analyzed. (A) OxLDL decreased co-immunoprecipitation of IκBα with both RelA and p50, and olaparib reversed the effect of oxLDL on the dissociation of IκBα with RelA and p50. …”
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MCP-1-deficiency decreased CD11c-expressing cells via impairing the production of ROS and decreased activation of PLCγ2, Akt, and ERK upon M-CSF stimulation in BMM.
Published 2013“…<p>BMMs from WT (open bar) and MCP-1-KO mice (oblique-lined bar) were incubated in the presence of M-CSF (30 ng/ml) with U73122 (10 µM), Akt inhibitor IV (0.3 µM), PD098059 (5 µM), DPI (50 nM), NAC (3 mM), or H<sub>2</sub>O<sub>2</sub> (300 µM) for 4 d (A). *, <i>P</i><0.05; ***, <i>P</i><0.001 compared with vehicle-treated WT cells. …”
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