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Differential regulation of axonal Prenyl-Cdc42 and RhoA mRNA levels (accompanying Fig 1).
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Growth-promoting and inhibiting stimuli regulate axonal levels of Prenyl-Cdc42 and RhoA mRNAs.
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Aggrecan-mediated increase in axonal KHSRP depletes axonal Prenyl-Cdc42 mRNA.
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datasheet1_Small Molecule Cjoc42 Improves Chemo-Sensitivity and Increases Levels of Tumor Suppressor Proteins in Hepatoblastoma Cells and in Mice by Inhibiting Oncogene Gankyrin.pdf
Published 2021“…Examination of Cjoc42 in WT mice showed that Cjoc42 is well tolerated without systemic toxicity, and levels of tumor suppressors CUGBP1, Rb, p53, C/EBPα, and HNF4α are increased by blocking their Gank-dependent degradation.…”
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Aggrecan triggered increase in KHSRP is limited to axons (accompanying Fig 6).
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Sequence alignment for vertebrate Prenyl-Cdc42 mRNA orthologs (accompanying Fig 3).
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ARE-binding protein KHSRP binds to non-localizing conserved region of Prenyl-Cdc42 3’UTR.
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