Muscle fiber hypertrophy in response to 6 weeks of high-volume resistance training in trained young men is largely attributed to sarcoplasmic hypertrophy by Cody T. Haun (4183900)

Polyamine depletion reroutes energy production to glycolysis. by Yazmin E. Cruz-Pulido (20285238)

Stochastic Formation of Quantum Defects in Carbon Nanotubes by Chen Ma (714759)

Stochastic Formation of Quantum Defects in Carbon Nanotubes by Chen Ma (714759)

Supplemental data on the effect of the M₂tail(368–466) fragment on mitochondrial function and cell metabolism. by Irene Fasciani (18462418)

Data Sheet 1_Diesel exhaust particles alter mitochondrial bioenergetics and cAMP producing capacity in human bronchial epithelial cells.pdf by Isabella Cattani-Cavalieri (14686957)

“…DEP significantly affected mRNA levels of exchange protein directly activated by cAMP-1 and -2 (Epac1, Epac2), appeared to increase Epac1 protein, but left phospho-PKA levels unhanged. …”

Image_2_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_1_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_1_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_3_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_3_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_2_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Impaired TFEB-mediated lysosomal biogenesis promotes the development of pancreatitis in mice and is associated with human pancreatitis by Shaogui Wang (310181)

Image_1_Fetal Programming of Renal Dysfunction and High Blood Pressure by Chronodisruption.JPEG by Natalia Mendez (311774)

“…At this age, CPS modified the expression of Nhe3, Ncc, Atp1a1, Nr3c1 (glucocorticoid receptor), and Nr3c2 (mineralocorticoid receptor); while Nkcc, Col3A1, and Opn were modified in the CPS 4%+NaCl group. …”