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Showing 1 - 14 results of 14 for search '(( (decrease OR increase) host resistance ) OR ( increases plaque resistance ))', query time: 0.10s Refine Results
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    Deletion of the Candida albicans PIR32 Results in Increased Virulence, Stress Response, and Upregulation of Cell Wall Chitin Deposition by Bahnan, Wael

    Published 2012
    “…Candida albicans is a common opportunistic pathogen that causes a wide variety of diseases in a human immunocompromised host leading to death. In a pathogen, cell wall proteins are important for stability as well as for acting as antigenic determinants and virulence factors. …”
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    A Severe Case of Overlap of Morphea and Eosinophilic Fasciitis after Burn Injuries by Hania Sami (21436013)

    Published 2024
    “…</p><h3>Conclusion</h3><p dir="ltr">Generalized morphea with concomitant EF indicates some degree of therapeutic resistance and poor prognosis with a low quality of life. …”
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    DNAJB3/HSP-40 cochaperone improves insulin signaling and enhances glucose uptake in vitro through JNK repression by Mohamed Abu-Farha (25322)

    Published 2015
    “…<p dir="ltr">Heat shock response (HSR) is an essential host-defense mechanism that is dysregulated in obesity-induced insulin resistance and type 2 diabetes (T2D). …”
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    Phenotypic and cell wall proteomic characterization of a DDR48 mutant candida albicans strain by El Khoury, Pamela

    Published 2019
    “…Cell wall proteins are the most important among these factors, being the first elements contacting the host. Ddr48 is a cell wall protein consisting of 212 amino acids. …”
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    Glycation in Alzheimer’s Disease and Type 2 Diabetes: The Prospect of Dual Drug Approaches for Therapeutic Interventions by Sama Ayoub (15430987)

    Published 2025
    “…Insulin resistance, a hallmark of T2DM, further complicates AD pathogenesis by promoting tau hyperphosphorylation and Aβ plaque accumulation. …”
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    Phenotypic and proteomic characterization of Dbr48, a candida albicans cell wall protein. (c2018) by Salameh, Carell C.

    Published 2018
    “…The most important virulence factors and antigenic determinants are the cell wall proteins as they are the first elements contacting the host. Ddr48 is a cell wall protein consisting of 212 amino acids. …”
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    masterThesis
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    Proteomic analyses of the cell walls of candida albicans PGA1 and PIR32 null mutants. (c2017) by Khoury, Pamela Milad El

    Published 2017
    “…The pga1 null strain was less adherent and virulent than the wild type strain and exhibited reductions in chitin deposition, biofilm formation, filamentation, and resistance to stress. The pir32 null strain on the other hand showed more pronounced phenotypes such as hyperfilamentation ability, and increased resistance to oxidative stress in part due to a two fold increase in chitin deposition. …”
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    masterThesis
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    Cost-effective microwave-assisted O3- type sodium-based layered oxide cathode materials for sodium-ion batteries by R.A. Harindi Gayara (17821406)

    Published 2023
    “…Finally, EIS analysis confirms the capacity fading of the developed materials during the cycling process is essentially due to an increase in the resistance with the increasing number of cycles due to the gradual thickening of formed SEI layer. …”
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    Comparative proteomic analysis of a Candida albicans DSE1 mutant under filamentous and non-filamentous conditions by Khalaf, Roy A.

    Published 2014
    “…The DSE1 mutant was shown to lack the chitin biosynthesis protein Chs5, explaining the previously observed decrease in chitin biosynthesis. The wild-type strain expressed Pra1, involved in pH response and zinc acquisition, Atg15, a lipase involved in virulence, and Sod1, required for oxidative stress tolerance, in addition to proteins involved in protein biosynthesis, explaining the increase in total protein content observed compared to the mutants strain. …”
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    Cytokine-Mediated Dysregulation of Signaling Pathways in the Pathogenesis of Multiple Myeloma by Sabah Akhtar (5809925)

    Published 2020
    “…Besides, these pathways also participate in developing resistance against the chemotherapeutic drugs in MM. …”
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