Responses of antennal neurons to sound. by Jonathan M. Blagburn (8259648)

“…(B) Responses (SEPs) of antennal auditory neurons to sound impulses of different magnitudes (sound level), from 7 different females at 10 days of age, showing a marked variation in peak SEP amplitude, from 0.5 to 1.7 mV. …”

Antibodies to PfRH5 and PfCyRPA modify temporal aspects of the pre- and post-invasion phases of <i>Plasmodium falciparum</i> into human RBCs. by Greta E. Weiss (16993535)

“…(F) Cy.007 antibodies decrease the numbers of echinocytosis events per egress and (G-H) R5.008 (pink) and Cy.009 (green) decrease the time from the end of invasion/deformation to echinocytosis. …”

Hemoglobin A1c Levels and Risk of Severe Hypoglycemia in Children and Young Adults with Type 1 Diabetes from Germany and Austria: A Trend Analysis in a Cohort of 37,539 Patients be... by Beate Karges (640991)

“…Based on advances in diabetes technology and pharmacotherapy, we hypothesized that the inverse association between severe hypoglycemia and HbA1c has decreased in recent years.…”

Levels of Ycg1 Limit Condensin Function during the Cell Cycle by Tyler W. Doughty (3147543)

“…Although several factors that promote maximal condensin activity during mitosis have been identified, the mechanisms that downregulate condensin activity during interphase are largely unknown. Here, we demonstrate that Ycg1, the Cap-G subunit of budding yeast condensin, is cell cycle-regulated with levels peaking in mitosis and decreasing as cells enter G1 phase. …”

OLM attenuates MTX effects on MPO and MDA. by Raimundo Fernandes de Araújo Júnior (429840)

OLM opposes MTX influence on cytokines. by Raimundo Fernandes de Araújo Júnior (429840)

Image_2_Identification of Two Missense Mutations in DUOX1 (p.R1307Q) and DUOXA1 (p.R56W) That Can Cause Congenital Hypothyroidism Through Impairing H2O2 Generation.tif by Shiguo Liu (180495)

“…DUOX2/DUOXA2 defects can cause congenital hypothyroidism (CH), but it is unknown whether DUOX1/DUOXA1 mutations can also cause CH.</p><p>Objective: We aimed to identify DUOX1/DUOXA1 mutations and explore their role in the development of CH by investigating their functional impacts on H₂O₂ generation.…”

Image_1_Identification of Two Missense Mutations in DUOX1 (p.R1307Q) and DUOXA1 (p.R56W) That Can Cause Congenital Hypothyroidism Through Impairing H2O2 Generation.tif by Shiguo Liu (180495)

“…DUOX2/DUOXA2 defects can cause congenital hypothyroidism (CH), but it is unknown whether DUOX1/DUOXA1 mutations can also cause CH.</p><p>Objective: We aimed to identify DUOX1/DUOXA1 mutations and explore their role in the development of CH by investigating their functional impacts on H₂O₂ generation.…”

Appearance and body color of <i>N</i>. <i>davidi</i> var. red. by Wei-Wei Hou (3405947)

BioID2-labelled putative interacting proteins. by Jan Pyrih (229124)

The two sides of public debt: Intergenerational altruism and burden shifting by Martin Fochmann (5693099)

“…However, we find considerably fewer intergenerational fairness concerns than one would expect on the basis of the behavioral and experimental literature. …”

Table_1_High-parameter phenotypic characterization reveals a subset of human Th17 cells that preferentially produce IL-17 against M. tuberculosis antigen.pdf by Paul Ogongo (5961161)

“…</p>Results<p>We identified two subsets of Th17 cells: subset 1 defined as CD4⁺Vα7.2⁻CD161⁺CD26⁺and subset 2 defined as CD4⁺Vα7.2⁻CCR6⁺CXCR3⁻cells of which subset 1 was significantly reduced in latent tuberculosis infection (LTBI) with HIV-ART, yet Mtb-responsive IL-17–producing CD4 T cells were preserved; we found that IL-17–producing CD4 T cells dominate the response to Mtb antigen but not cytomegalovirus (CMV) antigen or staphylococcal enterotoxin B (SEB), and tryptophan catabolism negatively correlates with both subset 1 and subset 2 Th17-cell frequencies.…”

Human papillomavirus type 16 E7 promotes cell viability and migration in cervical cancer by regulating the miR-23a/HOXC8 axis by Yahang Chen (17955991)

“…We used <i>in vitro</i> study to measure CC cell biological behaviours mediated by HPV16 E7, miR-23a and homeobox C8 (HOXC8). …”

Image_8_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.jpg by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Image_11_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.jpg by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Table_1_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.docx by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Table_3_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.docx by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Table_4_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.xls by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Image_6_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.jpg by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”

Table_2_Spliceostatin C, a component of a microbial bioherbicide, is a potent phytotoxin that inhibits the spliceosome.docx by Joanna Bajsa-Hirschel (3902302)

“…In silico binding studies showed that SPC binds to a pocket between the SF3B3 and PF5A of the spliceosome.…”