Search alternatives:
we decrease » _ decrease (Expand Search), nn decrease (Expand Search), teer decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
d decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
we decrease » _ decrease (Expand Search), nn decrease (Expand Search), teer decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
d decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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781
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782
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783
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784
Experiment 1: Comparison of effort and delay discounting.
Published 2015“…<b>C</b>, Individual and average fits of the sigmoidal winning model for effort and the hyperbolic winning model for delay. <b>D</b>, Mean squared error, indicating the goodness of fit of the two competing models for the effort and delay task. …”
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785
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786
CHIKV infection increases miR-146a expression and decreases TRAF6, IRAK1, and IRAK2 in primary human synovial fibroblasts.
Published 2014“…(C) Graph bars are showing densitometry analysis of TRAF6 normalized with housekeeping gene β-tubulin by ImageJ software. (D). Western blot analysis showing decrease in protein expression levels of IRAK 1 and IRAK 2 in CHIKV infected primary human synovial fibroblasts compared to uninfected controls. …”
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787
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788
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789
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790
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791
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792
miR-124 decreases cell proliferation in 2D and 3D cultures.
Published 2016“…Data from B are representative of 5 independent experiments; data from C are representative of 4 independent experiments, and data from D are representative of 2 independent experiments.…”
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793
Inhibition of mTOR pathway decreased the level of glycolytic metabolism in lung cancer cells.
Published 2015“…*<i>P</i><0.05; **<i>P</i><0.01; control versus AZD2014- or rapamycin-treated cells (see Fig 2d). Western blot analysis showed that AZD2014 and rapamycin decreased the level of PKM2 protein in a dose dependent manner. …”
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794
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795
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796
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797
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798
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799
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800