Cytotoxicity test for Aβ and AE subfractions. by Chien-Ning Huang (1739806)

Data_Sheet_1_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.pdf by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_1_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.pdf by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Training and salivary data set. by Davide Curzi (466778)

“…Moreover, this study investigated athletes’ hormonal response to a typical weekly conditioning session (5 days before match: MD-5), at T1 and T2, to detect changes in players’ recovery capability over time. …”

Timeline of the study. by Davide Curzi (466778)

“…Moreover, this study investigated athletes’ hormonal response to a typical weekly conditioning session (5 days before match: MD-5), at T1 and T2, to detect changes in players’ recovery capability over time. …”

The constitutively active form of insulin receptor and knockdown of FoxO block high temperature-mediated body fat loss. by Tucker Hopkins (20790529)

Foxo3a-dependent miR-633 regulates chemotherapeutic sensitivity in gastric cancer by targeting Fas-associated death domain by Xin Pang (249322)

A Model For A Narrow-Cut Fractionating Column by Farag, Hassan A.

“…A mathematical model was developed to simulate the operation of a narrow-cut fractionating column. …”
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β cell-specific deletion of HMG-CoA (3-hydroxy-3-methylglutaryl-coenzyme A) reductase causes overt diabetes due to reduction of β cell mass and impaired insulin secretion by Ada Admin (8524211)

La-induced differentiation delay in C2C12 cells is comparable to p38 MAPK-inhibition after 5 days of differentiation. by Lena Willkomm (298407)

Translocon inhibition partially prevents palmitate-induced reticular calcium depletion. by R. Cassel (849612)

Relative surface expression of Kv4.2^HA,EGFP. by Helena Prechtel (4811523)