CD5-CK2 Signaling Modulates Erk Activation and Thymocyte Survival by Carlos A. Mier-Aguilar (3605360)

“…Using a gene-targeted mouse in which the CD5-CK2BD is selectively ablated (CD5-ΔCK2BD), we determined that loss of function of CD5-CK2 signaling in a MHC-II selecting TCR transgenic (OT-II) mouse resulted in decrease in double positive (DP) thymocytes, which correlated with enhanced apoptosis. …”

Fig 5 - by Zhe Xu (127066)

Subjects:

Prorocentin-5: A Cytotoxic Polyketide from the Benthic Marine Dinoflagellate <i>Prorocentrum lima</i> by Yunseok Choi (5625245)

Subjects:

Decreased pulmonary cell inflammatory response in FI-A2 and G-A2 peptide vaccinated mice after RSV challenge. by Gertrud U. Rey (501694)

A Y45F mutation in hepatitis C virus NS4A causes a decrease in infectious viral titer. by Allison E. Roder (6312761)

Comparison of the decrease of CFU in CSF and blood between treatment groups within the first 6 h after treatment. by Luca G. Valente (12463057)

Fig 5 - by Thiago Bezerra Wanderley e Lima (14232350)

Stat1 activation is strongly decreased by MNV-S99 infection. by Sandra Niendorf (2832785)

Reduced expression of Tks5 leads to decreased growth of tumor cells <i>in vitro</i> and <i>in vivo</i>. by Barbara Blouw (715467)

Image_1_miR-144-5p and miR-451a Inhibit the Growth of Cholangiocarcinoma Cells Through Decreasing the Expression of ST8SIA4.tif by Wan Fu (3681799)

“…This study aimed to investigate the action mechanism of miR-144-5p and miR-451a in cholangiocarcinoma. We found that miR-144-5p and miR-451a were significantly decreased in cholangiocarcinoma patient samples compared to the adjacent normal bile duct samples. …”

Image_4_miR-144-5p and miR-451a Inhibit the Growth of Cholangiocarcinoma Cells Through Decreasing the Expression of ST8SIA4.tif by Wan Fu (3681799)

“…This study aimed to investigate the action mechanism of miR-144-5p and miR-451a in cholangiocarcinoma. We found that miR-144-5p and miR-451a were significantly decreased in cholangiocarcinoma patient samples compared to the adjacent normal bile duct samples. …”

Image_3_miR-144-5p and miR-451a Inhibit the Growth of Cholangiocarcinoma Cells Through Decreasing the Expression of ST8SIA4.tif by Wan Fu (3681799)

“…This study aimed to investigate the action mechanism of miR-144-5p and miR-451a in cholangiocarcinoma. We found that miR-144-5p and miR-451a were significantly decreased in cholangiocarcinoma patient samples compared to the adjacent normal bile duct samples. …”

Image_2_miR-144-5p and miR-451a Inhibit the Growth of Cholangiocarcinoma Cells Through Decreasing the Expression of ST8SIA4.tif by Wan Fu (3681799)

“…This study aimed to investigate the action mechanism of miR-144-5p and miR-451a in cholangiocarcinoma. We found that miR-144-5p and miR-451a were significantly decreased in cholangiocarcinoma patient samples compared to the adjacent normal bile duct samples. …”

Chemical Inhibition of eEF1A-1 Peptide Elongation Activity Decreases Palmitate-Induced Cell Death. by Alexandra M. Stoianov (759321)

Decreased tumorigenicity of A549 cells <i>in vivo</i> with reduced IQGAP3 expression. by Ying Yang (24175)

Genes whose expression was decreased in RASMC subjected to Na⁺,K⁺-ATPase inhibition. by Svetlana V. Koltsova (321381)

Fig 5 - by Zeenat A. Shyr (12107048)

VDR-dependent transcriptional activity of vitamin D derivatives in ATDC5 cells and analysis of the effect of binding to DBP. by Yoshihisa Hirota (616015)

“…<p>(A) Concentration dependence of the effects of vitamin D derivatives on VDR-dependent transcriptional activity of <i>CYP24A1</i> in ATDC5 cells. …”

AMPK phosphorylation and AC5 gene expression in hearts from sham and heart failure rats. by Anne Garnier (130125)

Subjects:

Data_Sheet_1_C5aR1 Activation Drives Early IFN-γ Production to Control Experimental Toxoplasma gondii Infection.DOCX by Daria Briukhovetska (9098294)

“…Our findings suggest a model in which the C5a/C5aR1 axis promotes IL-12 induction in splenic DCs that is critical for IFN-γ production from NK cells and subsequent iNOS expression in the brain as a critical mechanism to control acute T. gondii infection.…”