Showing 1,281 - 1,300 results of 148,443 for search '(( 2 a decrease ) OR ( 5 ((((wt decrease) OR (nn decrease))) OR (teer decrease)) ))', query time: 1.53s Refine Results
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    Downregulation of DOM decreases the abundance of PER and TIM. by Zhenxing Liu (399809)

    Published 2019
    “…<p><b>A.</b> Quantitative RT-PCR showing the expression of <i>clk</i>, <i>per</i> and <i>tim</i>. …”
  4. 1284

    Macrocyclic Metalloenediynes of Cu(II) and Zn(II):  A Thermal Reactivity Comparison by Tilak Chandra (1586797)

    Published 2001
    “…Rather, the Cu(II) complex exhibits the expected 9 °C decrease in the cyclization temperature (Cu(tact1:2) = 112 °C), whereas the temperature for the Zn(II) analogue increases by 15 °C (Zn(tact1:2) = 163 °C). …”
  5. 1285

    Preferential S-phase Est2 binding at short telomeres is decreased in <i>pif1-m2</i> cells. by Jane A. Phillips (729027)

    Published 2015
    “…(B) The lengths and identities of the telomeres being studied are indicated in the upper left corner of each graph. Binding of Est2-Myc at short telomere VII-L (top) was significantly decreased in <i>pif1-m2</i> (open circles) compared with WT cells (closed circles) during S phase (45 min, p = 0.025; 60 min, p = 0.0092; two-tailed unpaired <i>t</i>-tests). …”
  6. 1286

    The effects of GRK2, GRK2-K220R and β-arrestin 2 on GPR3 WT and ST/A activity and surface localization. by Katie M. Lowther (427408)

    Published 2013
    “…<p>GPR3-HA WT (<b>A</b>) and ST/A (<b>B</b>) were co-transfected with pcDNA, or GRK2, or GRK2 and β-arrestin-2, or GRK2-K220R, or GRK2-K220R and β-arr-2. …”
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    Identification of the DNA sequence within the mouse inhibin-α promoter required for Wt1A-dependent transcriptional activation. by Shao-Yang Ji (289371)

    Published 2013
    “…<p>A, TM4 cells were transiently transfected with luciferase reporter constructs bearing different lengths of the mouse Inhibin alpha promoter sequence, and Renilla luciferase reporter plasmids together with an empty plasmid (pCMV-tag2b) or Wt1A expression vector. …”
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