Table_1_Patient initiated follow-up in cancer patients: A systematic review.docx by Claire Newton (1773745)

“…</p>Objectives<p>To investigate the impact of PIFU on overall survival, progression free survival, patient satisfaction, psychological morbidity, specifically quality of life (QoL) and economic costs compared to hospital follow up (HFU), for any type of cancer.</p>Methods<p>We carried out a systematic review using five electronic databases: MEDLINE, CINAHL, EMBASE, PsycInfo and Cochrane Central Register of Controlled Trials. …”

Table_2_Patient initiated follow-up in cancer patients: A systematic review.docx by Claire Newton (1773745)

“…</p>Objectives<p>To investigate the impact of PIFU on overall survival, progression free survival, patient satisfaction, psychological morbidity, specifically quality of life (QoL) and economic costs compared to hospital follow up (HFU), for any type of cancer.</p>Methods<p>We carried out a systematic review using five electronic databases: MEDLINE, CINAHL, EMBASE, PsycInfo and Cochrane Central Register of Controlled Trials. …”

Bromodomain Inhibition Reveals FGF15/19 as a Target of Epigenetic Regulation and Metabolic Control by Chisayo Kozuka (656312)

Table_4_Patient initiated follow-up in cancer patients: A systematic review.docx by Claire Newton (1773745)

“…</p>Objectives<p>To investigate the impact of PIFU on overall survival, progression free survival, patient satisfaction, psychological morbidity, specifically quality of life (QoL) and economic costs compared to hospital follow up (HFU), for any type of cancer.</p>Methods<p>We carried out a systematic review using five electronic databases: MEDLINE, CINAHL, EMBASE, PsycInfo and Cochrane Central Register of Controlled Trials. …”

Structural and functional dissection reveals distinct roles of Ca²⁺-binding sites in the giant adhesin SiiE of <i>Salmonella enterica</i> by Britta Peters (4054456)

“…Amounts of secreted SiiE diminish with a decreasing number of intact Ca²⁺-binding sites. …”

Table_1_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.XLSX by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Table_6_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.xlsx by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Table_4_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.xlsx by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Table_7_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.xlsx by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Table_8_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.xlsx by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Table_3_Genome-Wide Identification of Rare and Common Variants Driving Triglyceride Levels in a Nevada Population.XLSX by Robert W. Read (6248582)

“…The study identified 549 significant single-variant associations (p < 8.75 × 10^–9), many in chromosome 11’s triglyceride hotspot: ZPR1, BUD13, APOC3, APOA5. A well-known protective loss-of-function variant in APOC3 (R19X) was associated with a 51% decrease in triglyceride levels in the cohort. …”

Image_1_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_1_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_3_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Image_3_Indomethacin Treatment Post-irradiation Improves Mouse Parotid Salivary Gland Function via Modulation of Prostaglandin E2 Signaling.TIFF by Kristy E. Gilman (11163543)

“…To define the signaling pathway whereby PGE₂ induces salivary gland dysfunction, primary parotid gland cells treated with PGE₂ have increased c-Jun N-terminal Kinase (JNK) activation and cell proliferation and reduced amylase levels and store-operated calcium entry (SOCE). …”

Table_1_Enterovirus 71 Antagonizes Antiviral Effects of Type III Interferon and Evades the Clearance of Intestinal Intraepithelial Lymphocytes.DOCX by Yuanmin Dong (12034109)

“…However, EV71 infection could markedly attenuate the antiviral responses of IFN-λ. Mechanistically, 2A protease (2Apro) and 3C protease (3Cpro) of EV71 inhibited the IFN-λ production and IFN-λ receptor expression and further decreased the response of IECs to IFN-λ. …”

Indoxyl sulfate altered differentiation marker expression in mouse podocytes. by Osamu Ichii (339354)

“…<p>The size of differentiated mouse podocytes decreased with indoxyl sulfate compared to dimethyl sulfoxide (DMSO) control; n = 3, mean ± SD (<b>a</b>). * denotes significant differences between the DMSO and indoxyl sulfate groups (<i>P</i><0.05). …”

Tumor targeting of Pt-Mal-LHRH attenuated breast cancer tumor growth. by Margaret Ndinguri (829018)

“…Tumor volume was measured over 2 wks and Pt-Mal-LHRH doses were found to significantly decrease tumor growth compared to carboplatin and control growth (2A). …”

Data_Sheet_1_Effects of intestinal microbes on rheumatic diseases: A bibliometric analysis.docx by Runzhi Huang (7428713)

“…The United States ranks at the top and has the broadest academic association with other countries. Five themes were identified, including the pivotal role of inflammation caused by intestinal bacteria in the rheumatic pathogenesis, the close relationship between rheumatic diseases and inflammatory bowel disease, immunoregulation mechanism as a mediator of the interaction between rheumatic diseases and gut flora, dysbiosis and decreased diversity in intestine of patients with rheumatic diseases, and the influence of oral flora on rheumatic diseases. …”

PCR targeting three regions within the prophage island found to be downregulated in Δ<i>mrvR</i>. by Allison N. Dammann (10271174)

“…The schematic shows the three amplified regions of the chromosome in red (<b>B</b>). qPCR validation of RNA-seq data showing decreased expression of RNA from regions 1 and 2 in the <i>mrvR</i> knockout compared to wild type (<b>C</b>).…”