Showing 1,101 - 1,120 results of 134,337 for search '(( 2 c decrease ) OR ( 5 ((((wt decrease) OR (we decrease))) OR (a decrease)) ))', query time: 2.16s Refine Results
  1. 1101

    Transaldolase deficiency alters mitochondrial morphology and decreases <i>in vivo</i> mitochondrial respiration. by Christopher F. Bennett (3875731)

    Published 2017
    “…The bottom panel consists of a max intensity projection of five z-slices to emphasize mitochondrial content in these cells. …”
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  11. 1111

    GluN2A(D731N) reduces the agonist potency. by Kai Gao (120150)

    Published 2017
    “…<p>(<b>A,B</b>) Representative TEVC recordings obtained from oocytes expressing WT GluN1/GluN2A (WT 2A) receptors (<b>A</b>) and GluN1/GluN2A(D731N) (2A-D731N) receptors (<b>B</b>) in which the currents were evoked by increasing concentrations (μM) of glutamate (in the presence of 100 μM glycine) at the holding potential of -40 mV. …”
  12. 1112

    GluN2A(D731N) enhances sensitivity to endogenous proton and zinc ions. by Kai Gao (120150)

    Published 2017
    “…Di-heteromeric (h2A-D731N), one-copy and two-copy mutant tri-heteromeric (D731N/2A and D731N/D731N) receptors show a decreased current ratio, indicating enhanced proton sensitivity. …”
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    Image_2_Decrease of GSK-3β Activity in the Anterior Cingulate Cortex of Shank3b−/− Mice Contributes to Synaptic and Social Deficiency.TIF by Mengmeng Wang (540017)

    Published 2019
    “…Shank3b mutation leads to a dramatic increase of pGSK-3β (Ser9), and decrease of pPSD95 (a substrate of GSK-3β) and GluR2. …”
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    NatB inactivation decreased the level of MAPK and proposed model of EGFR/MAPK signaling regulation by NatB and the N-end rule pathways. by Zhentao Sheng (107495)

    Published 2020
    “…In contrast, low NatB activity and low Ac-CoA levels will reduce the levels of Nt-acetylated Grb2/Drk, MAPK, and PP2AC, leading to reduced EGFR signaling due to decreased levels of Grb2/Drk and MAPK (positive components of the pathway) and increased levels of PP2AC (negative component of the pathway). …”
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