Metabolites with the top 5 positive and negative loadings (by absolute value) for PC1 and their respective pathway classification. by Manuel A. Cornejo (11636656)

Distribution of MAP-2+ dendrites in the spinal cord after T8 complete SCI. by Keun Woo Park (797704)

“…Scale bar, 250 μm. Quantification of MAP-2 immunoreactive intensity in spinal cords harvested at 1 (B-E) or 4 weeks (F-G) after SCI with Lenti-shSOCS3, Lenti-shSOCS3 #2, or Lenti-pGipz infection were analyzed in either gray (B, D, and F) or white matter (C, E, and G). …”

NO levels in aorta. by Haroldo A. Toque (447341)

Results of the evaluated LFAs and their performance parameters. by Ronald López (18098572)

Subjects:

Demographics, IgG anti-<i>T</i>. <i>cruzi</i> score, and location of residence of participants who provided samples used in this study. by Ronald López (18098572)

Subjects:

Sensitivity, specificity, and balanced accuracy for each index test (LFA); the dotted line indicates 95%. by Ronald López (18098572)

Subjects:

Proportion of time spent in the Start Room or Avoidance Room or showing a behaviour (Stimulus Contact) presented across stimuli and separately for piglets and gilts. by Sara Hintze (3216885)

Fig 6 - by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

Fig 2 - by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

Fig 1 - by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

Participant traits. by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

Fig 4 - by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

Fig 3 - by Matthew C. Kochem (18476069)

“…Plasma glucose, insulin, and glucagon were measured before, during, and after OGTTs up to 120 minutes post-prandially. We also assessed individual participants’ sweet taste responses to sucralose and their sensitivities to lactisole sweetness inhibition. …”

hsa-let-7e-5p acts as a downstream mediator of circ_0000006 and circ_0000160. by Yong Liu (6908)

Mean (±95% CI) of fold-change in monocular and binocular FMTs from smallest (7’) to largest (60’) target size obtained for mesopic and photopic conditions for central and parafovea... by C. Vijay Reena Durai (14498259)

Subjects:

Ursolic Acid Increases Skeletal Muscle and Brown Fat and Decreases Diet-Induced Obesity, Glucose Intolerance and Fatty Liver Disease by Steven D. Kunkel (156806)

(A): Orexin A inhibits propofol-induced neurite retraction by activation of phospholipase D. by Karin Björnström (118598)

“…OA increases the PKCε Ser⁷²⁹ phosphorylation compared to CCM, and this is reduced when PLD is blocked by FIPI, whereas propofol-treated cells showed a decrease in PKCε Ser⁷²⁹ phosphorylation that increased after FIPI treatment (n = 5). …”

Genetic polymorphisms in Nrf2 and FoxO1: implications for antioxidant enzyme activity in diabetes by Dipak A. Kadam (17052827)

Molecular signaling in response to diabetes. by Haroldo A. Toque (447341)

Inverse relationship between poison exon usage and expression of multiple sodium channels during mouse brain development. by Yuliya Voskobiynyk (9954619)

“…<p>(A) <i>Scn1a</i> transcripts including exon 20N are highly expressed in the developing mouse brain and decrease dramatically after birth (aqua bars), with a corresponding developmental increase in <i>Scn1a</i> expression (blue bars). …”