Showing 1,941 - 1,960 results of 123,408 for search '(( 2 d decrease ) OR ( 5 ((((wt decrease) OR (mean decrease))) OR (a decrease)) ))', query time: 1.64s Refine Results
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    Delphinidin alters VEGF-A splicing to increase VEGF-A<sub>165</sub>b and decrease total VEGF-A expression. by Megan Stevens (3964886)

    Published 2019
    “…<p><b>A)</b> Treatment of podocytes with delphinidin chloride (10 μg/ml) under normal glucose (NG; 5 mM glucose + 25 mM mannitol) and high glucose (HG; 30 mM glucose, 1 ng/ml TNFα, 1 ng/ml IL-6, and 100 nM insulin) for 48 hrs increased the protein expression of VEGF-A<sub>165</sub>b relative to total VEGF-A<sub>165</sub> (quantified in <b>B</b>; *p<0.05 vs NG, †p<0.05 vs HG; n = 3 biological repeats; One-way ANOVA with Bonferroni post-hoc test for comparison between pairs; <b>A</b>—the same blot was first probed with VEGF-A<sub>165</sub>b before stripping and reprobing with panVEGF-A). …”
  7. 1947

    Isoflurane decreased miR-214. by Hailiang Yan (284342)

    Published 2013
    “…(<b>E</b>) miR-214 decreased Bax expression in a dose-dependent manner. …”
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    Api5 depletion induces a decrease in E2F1-target gene mRNA levels without affecting E2F1 expression. by Marina Garcia-Jove Navarro (442768)

    Published 2013
    “…Api5, E2F1, cyclin E, cyclin A, cyclin D1, p16 INK4a, Cdk2 and Cdc25A relative mRNA levels were measured by quantitative PCR, using HPRT1 and GUSB mRNA levels as references. …”
  15. 1955
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    Activation of GluR5 increased neuronal excitability in interneruons but decreased excitability in pyramidal neurons. by Long-Jun Wu (47043)

    Published 2013
    “…Resting membrane potential for this neuron was −66.6 mV and the holding current was 66.9 pA. (H) Pooled data showed that ATPA (3 µM) increased firing rate of interneurons (n = 6) while decreased that of pyramidal neurons (n = 5). …”
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    Bortezomib in combination with the PLK1 inhibitor BI 2536 induces a significant downregulation of the total levels and phosphorylation of Bcr-Abl, a decrease of downstream phosphorylated STAT5 and a caspase-dependent cell death in TKIs-resistant and –sensitive K562 cell lines. by Octavian Bucur (470431)

    Published 2013
    “…The combined treatment resulted in a marked decrease of the total levels of Bcr-Abl, which correlates with a decrease in the phosphorylation of the downstream STAT5 protein, in both K562 and K562-R cells. β-Actin was used as an internal loading control. …”