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point decrease » point increase (Expand Search)
d decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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16021
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16022
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16023
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16024
FGF-9 Inhibits Infarct and Fibrosis in the Post-MI Diabetic Myocardium.
Published 2015“…<p><b>A-F</b>: Representative images of Masson’s trichrome stained sections are shown depicting infarct (<b>A-C</b>) and interstitial fibrosis (<b>D-F</b>) for control and experimental groups 2 weeks post-MI. …”
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16025
Socio-demographic factor for two cohorts aged 18-24 and 32-38 in 2016 in Gauteng.
Published 2025Subjects: -
16026
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16027
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16028
Sample Size for QoL tracking study for young people aged 18 to 24 in Gauteng, 2016 - 2024.
Published 2025Subjects: -
16029
Quality of Life changes between 2016 and 2024 for two age cohorts staying in Gauteng.
Published 2025Subjects: -
16030
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16031
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16032
PCAIs inhibit NCI-H23 cell line viability.
Published 2024“…Furthermore, 5 μM of NSL-YHJ-2-27 depleted the singly polyisoprenylated monomeric G-proteins RAC 1/2/3 and CDC42 by 77 and 76%, respectively. …”
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16033
PCAIs disrupt actin filaments in NCI-H23 cells.
Published 2024“…Furthermore, 5 μM of NSL-YHJ-2-27 depleted the singly polyisoprenylated monomeric G-proteins RAC 1/2/3 and CDC42 by 77 and 76%, respectively. …”
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16034
PCAIs suppress NCI-H23 cancer cell migration.
Published 2024“…Furthermore, 5 μM of NSL-YHJ-2-27 depleted the singly polyisoprenylated monomeric G-proteins RAC 1/2/3 and CDC42 by 77 and 76%, respectively. …”
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16035
PCAIs induce apoptosis in NCI-H23 cells.
Published 2024“…Furthermore, 5 μM of NSL-YHJ-2-27 depleted the singly polyisoprenylated monomeric G-proteins RAC 1/2/3 and CDC42 by 77 and 76%, respectively. …”
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16036
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16037
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16038
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16039
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16040
KIF5A-dependent axonal transport deficiency disrupts autophagic flux in trimethyltin chloride-induced neurotoxicity
Published 2024“…</p> <p><b>Abbreviations</b>: 3-MA: 3-methyladenine; AAV: adeno-associated virus; ACTB: actin beta; AGC: automatic gain control; ATG: autophagy-related; ATP6V0D1: ATPase H<sup>+</sup> transporting lysosomal V0 subunit D1; ATP6V1E1: ATPase H<sup>+</sup> transporting lysosomal V1 subunit E1; CA: cornu ammonis; CQ: chloroquine; CTSB: cathepsin B; CTSD: cathepsin D; DCTN1: dynactin subunit 1; DG: dentate gyrus; DYNLL1: dynein light chain LC8-type 1; FBS: fetal bovine serum; GABARAP: GABA type A receptor-associated protein; GABARAPL1: GABA type A receptor associated protein like 1; GABARAPL2: GABA type A receptor associated protein like 2; GAPDH: glyceraldehyde-3-phosphate dehydrogenase; IPA: Ingenuity Pathway Analysis; KEGG: Kyoto Encyclopedia of Genes and Genomes; KIF5A: kinesin family member 5A; LAMP: lysosomal-associated membrane protein; MAP1LC3B/LC3B: microtubule-associated protein 1 light chain 3 beta; NBR1: NBR1 autophagy cargo receptor; OPTN: optineurin; PBS: phosphate-buffered saline; PFA: paraformaldehyde; PIK3C3/VPS34: phosphatidylinositol 3-kinase catalytic subunit type 3; PRM: parallel reaction monitoring; siRNA: small interfering RNA; SQSTM1/p62: sequestosome 1; SYP: synaptophysin; TAX1BP1: Tax1 binding protein 1; TMT: trimethyltin chloride; TUB: tubulin.…”