Showing 11,901 - 11,920 results of 122,625 for search '(( 2 e decrease ) OR ( 5 ((ng decrease) OR (((mean decrease) OR (a decrease)))) ))', query time: 2.00s Refine Results
  1. 11901

    C5aR1 Mediates the Progression of Inflammatory Responses in the Brain of Rats in the Early Stage after Ischemia and Reperfusion by Yunwei Shi (10116568)

    Published 2021
    “…Further mechanism studies showed that inhibiting C5aR1 by PMX53 protected the rats from MCAO/R injury, decreased cell inflammation, and apoptosis via inhibiting the TLR4 and NF-κB signaling pathway and reducing the production of TNF-α, IL-1β, and IL-6 in MCAO/R rats. …”
  2. 11902

    C5aR1 Mediates the Progression of Inflammatory Responses in the Brain of Rats in the Early Stage after Ischemia and Reperfusion by Yunwei Shi (10116568)

    Published 2021
    “…Further mechanism studies showed that inhibiting C5aR1 by PMX53 protected the rats from MCAO/R injury, decreased cell inflammation, and apoptosis via inhibiting the TLR4 and NF-κB signaling pathway and reducing the production of TNF-α, IL-1β, and IL-6 in MCAO/R rats. …”
  3. 11903

    C5aR1 Mediates the Progression of Inflammatory Responses in the Brain of Rats in the Early Stage after Ischemia and Reperfusion by Yunwei Shi (10116568)

    Published 2021
    “…Further mechanism studies showed that inhibiting C5aR1 by PMX53 protected the rats from MCAO/R injury, decreased cell inflammation, and apoptosis via inhibiting the TLR4 and NF-κB signaling pathway and reducing the production of TNF-α, IL-1β, and IL-6 in MCAO/R rats. …”
  4. 11904

    C5aR1 Mediates the Progression of Inflammatory Responses in the Brain of Rats in the Early Stage after Ischemia and Reperfusion by Yunwei Shi (10116568)

    Published 2021
    “…Further mechanism studies showed that inhibiting C5aR1 by PMX53 protected the rats from MCAO/R injury, decreased cell inflammation, and apoptosis via inhibiting the TLR4 and NF-κB signaling pathway and reducing the production of TNF-α, IL-1β, and IL-6 in MCAO/R rats. …”
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  8. 11908

    Table_2_hnRNP E1 Regulates HPV16 Oncogene Expression and Inhibits Cervical Cancerization.docx by Li Song (34475)

    Published 2022
    “…Low expression of HPV16 E2, high expression of E6, and a low ratio of E2 to E6 could increase the risk of cervical lesions. hnRNP E1 expression was correlated with HPV16 oncogene expression. hnRNP E1-relevant genes were involved in the dopaminergic synapses, Wnt signaling pathway, gnRH secretion, and mTOR signaling pathway. hnRNP E1 significantly inhibited cell proliferation, induced apoptosis, arrested the cell cycle at the G0/G1 stage, and decreased HPV16 E6 expression. …”
  9. 11909

    Presentation_2_hnRNP E1 Regulates HPV16 Oncogene Expression and Inhibits Cervical Cancerization.pdf by Li Song (34475)

    Published 2022
    “…Low expression of HPV16 E2, high expression of E6, and a low ratio of E2 to E6 could increase the risk of cervical lesions. hnRNP E1 expression was correlated with HPV16 oncogene expression. hnRNP E1-relevant genes were involved in the dopaminergic synapses, Wnt signaling pathway, gnRH secretion, and mTOR signaling pathway. hnRNP E1 significantly inhibited cell proliferation, induced apoptosis, arrested the cell cycle at the G0/G1 stage, and decreased HPV16 E6 expression. …”
  10. 11910
  11. 11911

    Data_Sheet_5_Effects of APOE Genotype on Brain Proteomic Network and Cell Type Changes in Alzheimer's Disease.docx by Jingting Dai (6092432)

    Published 2018
    “…Individuals carrying the APOE E4 allele are at increased risk to develop AD compared to those carrying the more common E3 allele, whereas those carrying the E2 allele are at decreased risk for developing AD. …”
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  18. 11918
  19. 11919

    Lipidomic analysis of epidermis using LC-IMS-CID-MS reveals major changes in CKO// epidermis. by Kevin J. Mills (9270966)

    Published 2025
    “…Gray indicates lipids that are not significantly different in CKO vs K5Cre epidermis (p0.05 ANOVA). See (A) for lipid class nomenclature. …”
  20. 11920