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a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
point decrease » point increase (Expand Search)
e decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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18181
TPX2 selectively regulates the levels of H4K16ac during G1-phase.
Published 2014“…Note that the statistically significant (i.e. p<0.05) increase in γ-H2AX levels and decrease in H4K16ac levels upon TPX2 depletion is attenuated at the G1/S transition (i.e. 13 h after release). …”
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18182
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18183
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18184
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18185
Performance statistics on model simulations for river flow during calibration and validation.
Published 2024Subjects: -
18186
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18187
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18188
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18189
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18190
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18191
Image_1_Increased NOX2 expression in astrocytes leads to eNOS uncoupling through dihydrofolate reductase in endothelial cells after subarachnoid hemorrhage.TIF
Published 2023“…</p>Discussion<p>The increased level of NOX2 in astrocytes contributes to decreased DHFR in endothelial cells, thus aggravating eNOS uncoupling, which is an essential mechanism underlying acute vasoconstriction after SAH.…”
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18192
Image_3_Increased NOX2 expression in astrocytes leads to eNOS uncoupling through dihydrofolate reductase in endothelial cells after subarachnoid hemorrhage.TIF
Published 2023“…</p>Discussion<p>The increased level of NOX2 in astrocytes contributes to decreased DHFR in endothelial cells, thus aggravating eNOS uncoupling, which is an essential mechanism underlying acute vasoconstriction after SAH.…”
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18193
Table_1_Increased NOX2 expression in astrocytes leads to eNOS uncoupling through dihydrofolate reductase in endothelial cells after subarachnoid hemorrhage.docx
Published 2023“…</p>Discussion<p>The increased level of NOX2 in astrocytes contributes to decreased DHFR in endothelial cells, thus aggravating eNOS uncoupling, which is an essential mechanism underlying acute vasoconstriction after SAH.…”
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18194
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18195
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18196
AMPK activity and cell proliferation in HCC cell lines, PLC/PRF/5 and HepG2.
Published 2014“…(D): BrdU incorporation was significant decreased in both of PLC/PRF/5 and HepG2 cells by metformin treatment for 24 h (E): Metformin inhibited cell proliferation and the growth inhibition was rescued by AMPKα1 siRNA and compound C (an AMPK inhibitor) treatment (cell count method). …”
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18197
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18198
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18199
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18200