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2 mg » 2 mm (Expand Search), 5 mg (Expand Search), _ mg (Expand Search)
point decrease » point increase (Expand Search)
fold decrease » fold increase (Expand Search), fold increased (Expand Search)
mg decrease » _ decrease (Expand Search), nn decrease (Expand Search), we decrease (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
2 mg » 2 mm (Expand Search), 5 mg (Expand Search), _ mg (Expand Search)
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Damage accumulation decreases elongation rates and displaces growth equilibrium.
Published 2019“…<p>All panels depict MG1655 wild-type <i>E</i>. <i>coli</i>. (A) Exposure to phototoxic damage led to decreasing elongation rates in all treatment levels (length of exposure, every 2 min: 70 ms, 700 ms, 1 s, 1.5 s, 3 s). …”
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666
miR-200b is sufficient to decrease tumor growth.
Published 2013“…PC3-ctrl or PC3 miR-200b cells were seeded at 3000 cells per well in a 96-well plate. Absorbance was measured at indicated time points using a Biorad Model 680 microplate reader. …”
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667
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668
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669
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670
Acute JMV2959 treatment decreases alcohol preference in rats.
Published 2013“…<p>(A) Acute administration of the GHS-R1A antagonist JMV2959 (1 or 3 mg/kg) decreases the alcohol preference compared to vehicle at 1 hour time interval in rats voluntarily consuming alcohol for two and five months before the treatment. …”
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671
Acute JMV2959 treatment decreases alcohol intake in rats.
Published 2013“…<p>(A) The acute effects of the GHS-R1A antagonist JMV2959 (1 or 3 mg/kg) administration on alcohol intake was evaluated at 1 hour time interval after both two and five months of voluntary alcohol consumption. …”
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672
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673
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674
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675
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676
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677
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678
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679
Overexpression of Pax6 decreases Olig2 protein levels.
Published 2011“…(<b>a</b>) Oli-Neu cells were transfected with flag-tagged WT Pax6 (WT), Pax6(5a)R128C (128), or eGFP control vector (-). …”
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680
Image_2_EEG 40 Hz Coherence Decreases in REM Sleep and Ketamine Model of Psychosis.TIF
Published 2019“…Because of this fact, REM sleep is considered a natural model of psychosis. Ketamine is a non-competitive N-methyl-D-aspartate (NMDA) receptor antagonist that at sub-anesthetic dose induces psychotomimetic-like effects in humans and animals, and is employed as a pharmacological model of psychosis. …”