Search alternatives:
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
2 nn » 2 n (Expand Search), _ nn (Expand Search), k nn (Expand Search)
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
2 nn » 2 n (Expand Search), _ nn (Expand Search), k nn (Expand Search)
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11521
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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11522
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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11523
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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11524
Comparative Toxicity and Metabolism of N‑Acyl Homologues of Acetaminophen and Its Isomer 3′-Hydroxyacetanilide
Published 2016“…The hepatotoxicity of acetaminophen (APAP) is generally attributed to the formation of a reactive quinoneimine metabolite (NAPQI) that depletes glutathione and covalently binds to hepatocellular proteins. …”
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11525
Morphological changes in the testis and epididymis.
Published 2025“…B & F) There were no significant changes in the Vit versus control rats. C & G) PM2.5 rats exhibited sparse germinal layer in the seminiferous tubules, lumenal vacuolation and sperm shedding (black arrow) were observed, and the sperm quantity was significantly decreased. …”
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11526
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11527
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11528
Raw images of gels and blots.
Published 2025“…</p><p>Methods</p><p>In all, 32 male <i>SD</i> rats were randomized to a control cohort (normal saline), a Vit cohort (vitamin C at 100 mg/kg + vitamin E at 50 mg/kg), a PM2.5 cohort (PM2.5 10 mg/kg), and a PM2.5 + Vit cohort (PM2.5 exposure + vitamin C at 100 mg/kg + vitamin E at 50 mg/kg), with eight rats in each cohort. …”
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11529
Pregnancy rate of female rats in each group.
Published 2025“…</p><p>Methods</p><p>In all, 32 male <i>SD</i> rats were randomized to a control cohort (normal saline), a Vit cohort (vitamin C at 100 mg/kg + vitamin E at 50 mg/kg), a PM2.5 cohort (PM2.5 10 mg/kg), and a PM2.5 + Vit cohort (PM2.5 exposure + vitamin C at 100 mg/kg + vitamin E at 50 mg/kg), with eight rats in each cohort. …”
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11530
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11531
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11532
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11533
Table_1_Loss of function of VdDrs2, a P4-ATPase, impairs the toxin secretion and microsclerotia formation, and decreases the pathogenicity of Verticillium dahliae.DOCX
Published 2022“…In addition, the defect of VdDrs2 impaired the synthesis of melanin and the formation of microsclerotia, and decreased the sporulation of V. dahliae. Our data indicate a key role of P4 ATPases-associated vesicle transport in toxin secretion of disease fungi and support the importance of mycotoxins in the pathogenicity of V. dahliae.…”
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11534
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11535
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11536
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11537
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11538
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11539
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11540