Showing 17,541 - 17,560 results of 103,558 for search '(( 2 step decrease ) OR ( 5 ((point decrease) OR (((mean decrease) OR (a decrease)))) ))', query time: 1.84s Refine Results
  1. 17541

    Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

    Published 2022
    “…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”
  2. 17542

    Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

    Published 2022
    “…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”
  3. 17543

    Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

    Published 2022
    “…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”
  4. 17544

    Light-Powered Self-Sustained Oscillators of Graphene Oxide/Liquid Crystalline Network Composites Showing Amplitude and Frequency Superposition by Jianchuang Wang (2898131)

    Published 2022
    “…The GO/LCN composite film is prepared by the one-step polymerization of LC monomers, which favors a splay orientation in LC cells made by gluing together two glass sheets, one coated with photothermal agent GO and the other coated with a rubbed polyimide alignment layer. …”
  5. 17545

    Image_4_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  6. 17546

    DataSheet_1_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.docx by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  7. 17547

    Image_1_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  8. 17548

    Table_4_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.xlsx by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  9. 17549

    image_1_Complement Activation During Ischemia/Reperfusion Injury Induces Pericyte-to-Myofibroblast Transdifferentiation Regulating Peritubular Capillary Lumen Reduction Through pER... by Giuseppe Castellano (5258120)

    Published 2018
    “…Treatment by C1-inhibitor (C1-INH) significantly preserved the phenotype of pericytes maintaining microvascular density and capillary lumen area at tubulointerstitial level. In vitro, C5a transdifferentiated human pericytes in myofibroblasts, with increased αSMA expression in stress fibers, collagen I production, and decreased antifibrotic protein Id2. …”
  10. 17550

    image_2_Complement Activation During Ischemia/Reperfusion Injury Induces Pericyte-to-Myofibroblast Transdifferentiation Regulating Peritubular Capillary Lumen Reduction Through pER... by Giuseppe Castellano (5258120)

    Published 2018
    “…Treatment by C1-inhibitor (C1-INH) significantly preserved the phenotype of pericytes maintaining microvascular density and capillary lumen area at tubulointerstitial level. In vitro, C5a transdifferentiated human pericytes in myofibroblasts, with increased αSMA expression in stress fibers, collagen I production, and decreased antifibrotic protein Id2. …”
  11. 17551

    Image_7_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  12. 17552

    image_3_Complement Activation During Ischemia/Reperfusion Injury Induces Pericyte-to-Myofibroblast Transdifferentiation Regulating Peritubular Capillary Lumen Reduction Through pER... by Giuseppe Castellano (5258120)

    Published 2018
    “…Treatment by C1-inhibitor (C1-INH) significantly preserved the phenotype of pericytes maintaining microvascular density and capillary lumen area at tubulointerstitial level. In vitro, C5a transdifferentiated human pericytes in myofibroblasts, with increased αSMA expression in stress fibers, collagen I production, and decreased antifibrotic protein Id2. …”
  13. 17553

    Image_6_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  14. 17554

    Image_2_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  15. 17555

    Table_1_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.xlsx by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  16. 17556

    Table_3_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.xlsx by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  17. 17557

    Table_2_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.xlsx by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  18. 17558

    image_4_Complement Activation During Ischemia/Reperfusion Injury Induces Pericyte-to-Myofibroblast Transdifferentiation Regulating Peritubular Capillary Lumen Reduction Through pER... by Giuseppe Castellano (5258120)

    Published 2018
    “…Treatment by C1-inhibitor (C1-INH) significantly preserved the phenotype of pericytes maintaining microvascular density and capillary lumen area at tubulointerstitial level. In vitro, C5a transdifferentiated human pericytes in myofibroblasts, with increased αSMA expression in stress fibers, collagen I production, and decreased antifibrotic protein Id2. …”
  19. 17559

    Image_3_m6A mRNA Methylation Regulates Epithelial Innate Antimicrobial Defense Against Cryptosporidial Infection.tif by Zijie Xia (3772114)

    Published 2021
    “…Altered m<sup>6</sup>A methylation in mRNAs in intestinal epithelial cells following C. parvum infection is associated with downregulation of alpha-ketoglutarate-dependent dioxygenase alkB homolog 5 and the fat mass and obesity-associated protein with the involvement of NF-кB signaling. …”
  20. 17560

    DataSheet_1_Targeting Glutamine Metabolism Ameliorates Autoimmune Hepatitis via Inhibiting T Cell Activation and Differentiation.docx by Qiang Yu (5487)

    Published 2022
    “…Besides, we demonstrated that glutamine metabolism blocking inhibited T cells activation and differentiation through decreasing the mRNA expression of amino acid transporter solute carrier family 7 member 5 (SLC7A5) and mitigating the activation of mTOR signaling.…”