SMC-specific knockout of Klf5 (smcKlf5^−/−) exacerbates the progression of Ang II–induced AAA by facilitating ROS formation. by Dong Ma (584680)

Subjects:

The correlation of the mitochondrial dynamics–related genes with Klf5 in mouse VSMCs. by Dong Ma (584680)

Subjects:

Numerical raw data. by Dong Ma (584680)

Subjects:

The primers for real-time PCR. by Dong Ma (584680)

Subjects:

ERK signaling and expression of β-arrestin2 in Ang II–induced mouse AAA models as well as in WT or Klf5^−/− VSMCs. by Dong Ma (584680)

Subjects:

Demographic and clinical characteristics of patients with AAA. by Dong Ma (584680)

Subjects:

The expression of eIF5a, Fis1, Pink1, Drp1, Mfn1, and Mtfr1 and the analysis of mitochondrial morphology. by Dong Ma (584680)

Subjects:

The effect of ML264 (Klf5 inhibitor) on aneurysm formation induced by Ang II infusion in the early and late stages. by Dong Ma (584680)

Subjects:

Cardiac function assessed by echocardiography in Ang II–infused young (3 months) or old (18 months) WT and smcKlf5^−/− mice. by Dong Ma (584680)

Subjects:

Klf5 deficiency leads to mitochondrial dyfunction in VSMCs. by Dong Ma (584680)

Subjects:

Klf5 deficiency in mouse VSMCs leads to mitochondrial fission. by Dong Ma (584680)

Subjects:

ATP assay. by Dong Ma (584680)

Subjects:

Representative TUNEL- and DAPI-stained sections from the abdominal aortas of young and old WT and smcKlf5^−/− mice following 28 days of Ang II infusion. by Dong Ma (584680)

Subjects:

eIF5a affects the expression of mitochondrial dynamics–related genes. by Dong Ma (584680)

Subjects:

Young (3 months) or old (18 months) WT and smcKlf5^−/− mice were infused with Ang II for 28 days. by Dong Ma (584680)

Subjects:

Klf5 down-regulation leads to human VSMC senescence. by Dong Ma (584680)

Subjects:

Demographic information of survey participants. by Katie J. Shillington (16557695)

Subjects:

The spectral sensitivity of European seabass fed a 5% taurine diet. by Richard W. Brill (2337940)

Decreased Sho levels in ScN2a-Sho cells. by Joel C. Watts (195735)

“…(<b>C</b>) Quantification of Sho levels in ScN2a-Sho-1 (<i>n</i> = 10) and ScN2a-Sho-2 (<i>n</i> = 5) subclones revealed a significant decrease of 40–45% compared to the uninfected parental cell line (<i>n</i> = 15) (***<i>P</i><0.001).…”

Stochastic simulations show an initial decrease in viral load down to a relatively low level could lead to stochastic extinction, which cannot be explained by deterministic models. by Pengxing Cao (615266)