eIF5a interacts with Mfn1 and preserves mitochondrial integrity. by Dong Ma (584680)

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Inhibition of Ric-8A or Gα_i activity decreases the recruitment of PtdIns(3)P sensor. by Cedric Boularan (269965)

eIF5a affects the expression of mitochondrial dynamics–related genes. by Dong Ma (584680)

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Klf5 deficiency in mouse VSMCs leads to mitochondrial fission. by Dong Ma (584680)

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Mitochondrial morphology assay. by Dong Ma (584680)

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Analysis of relative fluorescence intensity. by Dong Ma (584680)

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ATP assay. by Dong Ma (584680)

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POS treatment decreases ROS level and upregulates mitochondrial biogenesis in ARPE-19 cells through PGC-1α. by Murilo F. Roggia (755997)

NAC suppresses Ang II–induced mitochondrial fission in mouse VSMCs. by Dong Ma (584680)

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Photostimulation of DRN 5-HT neurons <i>in vivo</i>. by Guillaume P. Dugué (620333)

Effect of eIF5a and Mfn1 expression on mitochondrial morphology. by Dong Ma (584680)

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The expression of eIF5a, Fis1, Pink1, Drp1, Mfn1, and Mtfr1 and the analysis of mitochondrial morphology. by Dong Ma (584680)

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The correlation of the mitochondrial dynamics–related genes with Klf5 in mouse VSMCs. by Dong Ma (584680)

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(+)-Usnic acid decreases mRNA level of downstream target genes of β-catenin/LEF and c-jun/AP-1. by Yi Yang (116183)

Low oscillatory shar stress <i>Arf6</i> heterozygosity decreases Arf6 expression. by Venkateswara R. Gogulamudi (15447428)

Alkaline Phosphatase, Soluble Extracellular Adenine Nucleotides, and Adenosine Production after Infant Cardiopulmonary Bypass by Jesse A. Davidson (3158913)

“…Inhibition of alkaline phosphatase led to a marked decrease in 13C5-adenosine production (11.9μmol/L vs 2.7μmol/L; p<0.0001). …”

Changes associated with kidney fibrosis and effects of MN705 treatment on TGF- by Wencheng Jin (13136262)

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Changes related to kidney staining in UUO mouse model. by Wencheng Jin (13136262)

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Changes associated with kidney fibrosis in TGF- by Wencheng Jin (13136262)

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Apoptosis inhibitor of macrophage depletion decreased M1 macrophage accumulation and the incidence of cardiac rupture after myocardial infarction in mice by Shohei Ishikawa (4583068)

“…</p><p>Conclusions</p><p>These findings suggest that AIM depletion decreases the levels of M1 macrophages, which are a potent source of MMP-2 and 9, in the infarcted myocardium in the acute phase after MI by promoting macrophage apoptosis, and leads to a decrease in the incidence of cardiac rupture and improvements in survival rates.…”