Generating Oscillatory Behavior by Applying a Magnetic Field during Electrocatalytic Oxidation of Glycerol by Rui Gao (247606)

Subjects:

Generating Oscillatory Behavior by Applying a Magnetic Field during Electrocatalytic Oxidation of Glycerol by Rui Gao (247606)

Subjects:

Generating Oscillatory Behavior by Applying a Magnetic Field during Electrocatalytic Oxidation of Glycerol by Rui Gao (247606)

Subjects:

Strains used in this study. by Xin Zhou (54275)

“…Dysfunction of <i>ERG251</i>, together with FLC exposure, resulted in decreased accumulation of the toxic sterol (14-ɑ-methylergosta-8,24(28)-dien-3β,6α-diol) and increased accumulation of non-toxic alternative sterols. …”

Primers used in this study. by Xin Zhou (54275)

“…Dysfunction of <i>ERG251</i>, together with FLC exposure, resulted in decreased accumulation of the toxic sterol (14-ɑ-methylergosta-8,24(28)-dien-3β,6α-diol) and increased accumulation of non-toxic alternative sterols. …”

The point mutation of <i>ERG251</i> leads to the partial dysfunction of <i>ERG251</i> causing acquisition of azole tolerance. by Xin Zhou (54275)

Drp1^SD overexpression in Opa1 or Marf depleted type II NBs rescues the reduced mitochondrial membrane potential. by Dnyanesh Dubal (12085942)

“…<p>A, B: Representative images showing decreased TMRM intensity in <i>opa1</i> RNAi and <i>marf</i> RNAi expressing type II NBs. …”

<i>ERG251</i> SNPs from all FLC-evolved strains. by Xin Zhou (54275)

“…Dysfunction of <i>ERG251</i>, together with FLC exposure, resulted in decreased accumulation of the toxic sterol (14-ɑ-methylergosta-8,24(28)-dien-3β,6α-diol) and increased accumulation of non-toxic alternative sterols. …”

A nonsense mutation in <i>anmK</i> contributes to decreased growth of AC811 in Arabidopsis leaves. by Megan R. O’Malley (7485323)

Notch signaling regulates fused mitochondrial morphology in INPs and muscle cells. by Dnyanesh Dubal (12085942)

Expression of <i>uvrC</i> is decreased in AC811 but not in Δ<i>gacA</i>-1. by Megan R. O’Malley (7485323)

Analysis of mitochondrial morphology in Notch mutants and Notch signaling in Opa1 depleted type II NB lineages. by Dnyanesh Dubal (12085942)

Analysis of change in mature INPs, GMCs and neurons in the type II NB lineage on depletion of mitochondrial morphology proteins. by Dnyanesh Dubal (12085942)

Drp1^SD overexpression partially alleviates the differentiation defect seen in <i>notch</i> RNAi expressing type II NB lineages. by Dnyanesh Dubal (12085942)

Expression of mRFP does not perturb the severity of <i>opa1</i> RNAi and <i>marf</i> RNAi phenotypes. by Dnyanesh Dubal (12085942)

Downregulation of Opa1 and Marf does affect type II NB numbers but affect differentiated cells in lineage. by Dnyanesh Dubal (12085942)

Analysis of proliferation in NBs depleted of Opa1 and Marf. by Dnyanesh Dubal (12085942)

Depletion of Opa1 and Marf does not cause increase in the cleaved Caspase 3 levels and apoptosis in the brain. by Dnyanesh Dubal (12085942)

Depletion of mitochondrial fusion proteins Opa1 and Marf result in mitochondrial fragmentation. by Dnyanesh Dubal (12085942)

<i>WDR5</i> Expression Is Prognostic of Breast Cancer Outcome by Xiaofeng Dai (793874)

“…<div><p>WDR5 is a core component of the human mixed lineage leukemia-2 complex, which plays central roles in ER positive tumour cells and is a major driver of androgen-dependent prostate cancer cell proliferation. …”