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64661
GABA<sub>B</sub> receptor knockdown affects levels of trehalose and lipid at starvation.
Published 2010“…<p><b>A</b>. Trehalose levels were determined in fed flies and flies starved for 5 and 12 h (0, 5, 12 in bars) in flies with GABA<sub>B</sub> receptor knockdown in IPCs (Dilp2-GBRi) and parental controls. …”
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64662
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64663
Table_1_Multi-Domain Touchscreen-Based Cognitive Assessment of C57BL/6J Female Mice Shows Whole-Body Exposure to 56Fe Particle Space Radiation in Maturity Improves Discrimination L...
Published 2021“…As there are conflicting results on whether the female rodent brain is preferentially injured by or resistant to charged particle exposure, and as the proportion of female vs. male astronauts is increasing, further study on how charged particles influence the touchscreen cognitive performance of female mice is warranted. We hypothesized that, similar to mature male mice, mature female C57BL/6J mice exposed to fractionated whole-body <sup>56</sup>Fe irradiation (3 × 6.7cGy <sup>56</sup>Fe over 5 days, 600 MeV/n) would improve performance vs. …”
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64664
Data_Sheet_1_Genome-Wide Characterization of Ascorbate Peroxidase Gene Family in Peanut (Arachis hypogea L.) Revealed Their Crucial Role in Growth and Multiple Stress Tolerance.ZIP
Published 2022“…<p>Ascorbate peroxidase (APX), an important antioxidant enzyme, plays a significant role in ROS scavenging by catalyzing the decrease of hydrogen peroxide under various environmental stresses. …”
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64665
Data_Sheet_1_Genome-Wide Characterization of Ascorbate Peroxidase Gene Family in Peanut (Arachis hypogea L.) Revealed Their Crucial Role in Growth and Multiple Stress Tolerance.ZIP
Published 2022“…<p>Ascorbate peroxidase (APX), an important antioxidant enzyme, plays a significant role in ROS scavenging by catalyzing the decrease of hydrogen peroxide under various environmental stresses. …”
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64666
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64667
<i>In vitro</i> ASC withstand hyperglycemic stress with minimal apoptosis and increase retinal endothelial survival.
Published 2014“…<p>(A). Number of viable cells at normal (5.5 mM) or high glucose concentrations did not change at 72hrs as evidenced from the quantity of formazan product measured at 490 nm, is directly proportional to the number of living cells in culture. …”
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64668
Effects of CB<sub>1</sub> inhibition on signal transduction, p65-level, cell cycle profile and apoptotic populations in L428 cells.
Published 2013“…AM251-treatment resulted in higher amounts of cleaved of caspase-3 (cleaved C-3) accompanied by a decrease of full length caspase-3 (C-3). Values represent means ± SD of 3 independent experiments.…”
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64669
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64670
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64671
Genetic and Metabolomic Dissection of the Ergothioneine and Selenoneine Biosynthetic Pathway in the Fission Yeast, <i>S. pombe</i>, and Construction of an Overproduction System
Published 2014“…In this study we report the genes forming the two-step ergothioneine biosynthetic pathway in the fission yeast, <i>Schizosaccharomyces pombe</i>. …”
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64672
Comprehensive Functional Annotation of 77 Prostate Cancer Risk Loci
Published 2014“…To aid the identification of these enhancers, we performed genomewide ChIP-seq for H3K27-acetylation, a mark of actively engaged enhancers, as well as the transcription factor TCF7L2. We analyzed in depth three variants in risk enhancers, two of which show significantly altered androgen sensitivity in LNCaP cells. …”
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64673
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64674
Image_3_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg
Published 2022“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64675
Image_4_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG
Published 2020“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64676
Image_1_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG
Published 2020“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64677
Image_3_RETRACTED: Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg
Published 2024“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64678
Image_1_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG
Published 2022“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64679
Image_4_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG
Published 2022“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”
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64680
Image_4_RETRACTED: Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG
Published 2024“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”