Image 3_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 7_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 2_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 1_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 8_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 9_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 7_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Image 4_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.tif by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 1_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 6_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xls by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 2_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.docx by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Table 4_Integrative RNA-seq and CLIP-seq analysis reveals hnRNP-F regulation of TNFα/NFκB signaling in high-glucose conditions.xlsx by Lan Wang (85307)

“…</p>Results<p>In this study, RNA sequencing (RNA-seq) was utilized to demonstrate that the upregulation of hnRNP-F in HK-2 cells cultured under high-glucose conditions resulted in a substantial decrease in the expression of genes associated with the inflammatory response and suppression of the TNFα-NFκB signaling pathway. …”

Glucagon Clearance is Preserved in Type 2 Diabetes by Magnus F.G. Grøndahl (11576700)

Formation of Antiferromagnetically Coupled C₆₀^•- and Diamagnetic (C₇₀^-)₂ Dimers in Ionic Complexes of Fullerenes with (MDABCO... by Dmitri V. Konarev (1405348)

“…IR− and UV−vis-NIR spectra of <b>1</b>−<b>6</b> justified the formation of C₆₀^•- in <b>1</b>−<b>4</b> and single-bonded (C₇₀^-)₂ dimers in <b>5</b> and <b>6</b>. Co and Mn atoms are six-coordinated in the (MDABCO⁺)₂·M^IITPP units with relatively long M−N bonds of 2.475(2), 2.553(2), and 2.511(3) Å for <b>2,</b> <b>3</b>, and <b>5</b>, respectively. …”

Formation of Antiferromagnetically Coupled C₆₀^•- and Diamagnetic (C₇₀^-)₂ Dimers in Ionic Complexes of Fullerenes with (MDABCO... by Dmitri V. Konarev (1405348)

“…IR− and UV−vis-NIR spectra of <b>1</b>−<b>6</b> justified the formation of C₆₀^•- in <b>1</b>−<b>4</b> and single-bonded (C₇₀^-)₂ dimers in <b>5</b> and <b>6</b>. Co and Mn atoms are six-coordinated in the (MDABCO⁺)₂·M^IITPP units with relatively long M−N bonds of 2.475(2), 2.553(2), and 2.511(3) Å for <b>2,</b> <b>3</b>, and <b>5</b>, respectively. …”

Formation of Antiferromagnetically Coupled C₆₀^•- and Diamagnetic (C₇₀^-)₂ Dimers in Ionic Complexes of Fullerenes with (MDABCO... by Dmitri V. Konarev (1405348)

“…IR− and UV−vis-NIR spectra of <b>1</b>−<b>6</b> justified the formation of C₆₀^•- in <b>1</b>−<b>4</b> and single-bonded (C₇₀^-)₂ dimers in <b>5</b> and <b>6</b>. Co and Mn atoms are six-coordinated in the (MDABCO⁺)₂·M^IITPP units with relatively long M−N bonds of 2.475(2), 2.553(2), and 2.511(3) Å for <b>2,</b> <b>3</b>, and <b>5</b>, respectively. …”

Developmental trends in flux partitioning between successive internodes. by Yun Lee (121214)

Intrachromatid and intersister (IC/IS) bias in <i>URA3</i> inactivation. by James D. Cauwood (481139)

“…Indicated are the migrating positions for WT, INT, and PO fragments described in panel A. <b>C</b>, <b>D.</b> Representative images of Southern Blot analyses on 5FOA^R colonies that arose in YPD or HU. …”

Image_2.tif by Yean K. Yong (727047)

“…We report that decrease of TCR iVα7.2⁺ MAIT cells in the peripheral blood and their functions were seemingly impaired in chronic HBV-infected patients likely because of the increased expression of PD-1.…”

Image_3.tif by Yean K. Yong (727047)

“…We report that decrease of TCR iVα7.2⁺ MAIT cells in the peripheral blood and their functions were seemingly impaired in chronic HBV-infected patients likely because of the increased expression of PD-1.…”