Table_2_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.XLSX by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Data Sheet 1_Patient-centered modeling of the breast biopsy experience.pdf by Isabel Nieto-Alvarez (22425466)

“…Centering each individual's procedure time at her first local anesthesia (LA) revealed a significant (βt2p = 5.43e⁻⁰⁶) temporal pattern in anxiety, which increased until LA and decreased afterwards. …”

Stochastic Formation of Quantum Defects in Carbon Nanotubes by Chen Ma (714759)

“…For bulk samples, the bandgap emission (E₁₁) of monochiral (6,5)-SWCNTs decreases, and the defect-related emission feature (E₁₁*) increases with diazonium salt concentration and represents a proxy for the defect number. …”

Image_3_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Elk1 affects katanin and spastin proteins via differential transcriptional and post-transcriptional regulations by Dolunay Kelle (6388055)

“…Even though severing mechanism is relatively well-studied, there are limited studies for the transcriptional regulation of microtubule severing proteins. In this study, we identified the main regulatory region of <i>KATNA1</i> gene encoding katanin-p60 as 5’ UTR, which has a key role for its expression, and showed Elk1 binding to <i>KATNA1</i>. …”

Table_1_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.XLSX by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Table_1_Juvenility and Vegetative Phase Transition in Tropical/Subtropical Tree Crops.DOCX by Muhammad Umair Ahsan (6792440)

“…We observed that miR156 expression decreases as these trees age and can potentially be used as a juvenility marker. …”

Image_1_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Data_Sheet_1_Enterovirus 71 Suppresses miR-17-92 Cluster Through Up-Regulating Methylation of the miRNA Promoter.docx by Yuxuan Fu (4437475)

“…Overexpression of hsa-miR-17∼92 inhibited, while inhibition of endogenous hsa-miR-17∼92 facilitated EV71 replication. We identified two sequences located at nt 3024 to 3038 and nt 2838 to 2862 of the EV71 (strain FY0805) genome as potential targets for hsa-miR-17-5p and miR-19a/b, respectively, which were validated by luciferase reporter assays and Western blot. …”

Table_1_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.XLSX by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Image_3_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Image_4_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Supplementary Material for: Persistently Altered Metabolic Phenotype following Perinatal Excitotoxic Brain Injury by Blaise B.J. (4019153)

“…Specifically, we identified increases in the amino acids glutamine, proline, serine, threonine, tryptophan, valine, and the sphingolipid SM C26:1, and decreases in the glycerophospholipids, i.e., the arachidonic acid-containing phosphatidylcholine (PC aa) C30:2 and the PC aa C32:3. …”

Image_2_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Stochastic Formation of Quantum Defects in Carbon Nanotubes by Chen Ma (714759)

“…For bulk samples, the bandgap emission (E₁₁) of monochiral (6,5)-SWCNTs decreases, and the defect-related emission feature (E₁₁*) increases with diazonium salt concentration and represents a proxy for the defect number. …”

Image_2_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.TIFF by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

Table_2_Differential Profile of Systemic Extracellular Vesicles From Sporadic and Familial Alzheimer’s Disease Leads to Neuroglial and Endothelial Cell Degeneration.XLSX by Juan Villar-Vesga (4900768)

“…We found increase CLN-5 spots with vesicular morphology in the abluminal portion of vessels from SAD patients. …”

HIRA Is Required for Heart Development and Directly Regulates <i>Tnni2</i> and <i>Tnnt3</i> by Daniel Dilg (2940339)

“…We found that HIRA binds GAGA rich DNA loci in the embryonic heart, and in particular a previously described enhancer of <i>Tnni2/Tnnt3</i> (<i>TTe</i>) bound by the transcription factor NKX2.5. …”

Simulation of hypGOI expression study in VK2/E6E7 cells treated with <i>Lactobacillus acidophilus</i> NCFM. by Annette V. Jacobsen (677206)

“…The most stable gene assessed (<i>RPLP0</i>) was used as the standard for the target fold changes, which were set at two-fold decrease (A), two-fold increase (B), five-fold decrease (C), and five-fold increase (D). …”

Simulation of hypGOI expression study in HT-29 cells treated with <i>Lactobacillus acidophilus</i> NCFM. by Annette V. Jacobsen (677206)

“…The most stable gene assessed (<i>PGK1</i>) was used as the standard for the target fold changes, which were set at two-fold decrease (A), two-fold increase (B), five-fold decrease (C), and five-fold increase (D). …”