Image1_v1_Treatment of Spleen-Deficiency Syndrome With Atractyloside A From Bran-Processed Atractylodes lancea by Protection of the Intestinal Mucosal Barrier.TIF by Jiyuan Tu (572853)

Image1_v1_Treatment of Spleen-Deficiency Syndrome With Atractyloside A From Bran-Processed Atractylodes lancea by Protection of the Intestinal Mucosal Barrier.TIF by Jiyuan Tu (572853)

Table1_v1_Treatment of Spleen-Deficiency Syndrome With Atractyloside A From Bran-Processed Atractylodes lancea by Protection of the Intestinal Mucosal Barrier.DOCX by Jiyuan Tu (572853)

Table1_v1_Treatment of Spleen-Deficiency Syndrome With Atractyloside A From Bran-Processed Atractylodes lancea by Protection of the Intestinal Mucosal Barrier.DOCX by Jiyuan Tu (572853)

Comparison of mitochondrial respiration of HL-1 cells exposed to LG, NG, HG and IHG between short- (12h) and long-time (72h) glucose treatment. by Patrick Mordel (17020879)

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“…1,3-bis(4-nitrophenyl)urea (<b>1</b>) interacts through hydrogen bonding with a variety of oxoanions in an MeCN solution to give bright yellow 1:1 complexes, whose stability decreases with the decreasing basicity of the anion (CH₃COO^- > C₆H₅COO^- > H₂PO₄^- > NO₂^- > HSO₄^- > NO₃^-). …”

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Detection of oxidized proteins by oxyblot in HL-1 cells exposed for 12 or 72h to LG, NG, HG or IHG. by Patrick Mordel (17020879)

Comparison of ATP production in HL-1 cells exposed to LG, NG, HG and IHG between short- (12h) and long-time (72h) glucose treatment. by Patrick Mordel (17020879)

Data_Sheet_5_Suppression of hnRNP A1 binding to HK1 RNA leads to glycolytic dysfunction in Alzheimer’s disease models.zip by Xin-Hao Ji (16910400)

Omeprazole (Ome) decreases IL-4-stimulated RNA Pol II binding to the endogenous eotaxin-3 promoter in (A) EoE1-T and (B) EoE2-T cells. by Xi Zhang (83736)

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Hypoxic MSCs stimulate regulatory T cell proliferation via paracrine mechanisms. by Rohini L. Kadle (4919335)