Image_2_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.JPEG by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Image_7_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.PNG by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Image_1_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.JPEG by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Image_4_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.JPEG by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Table_1_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.DOCX by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Image_3_Study of CD27, CD38, HLA-DR and Ki-67 immune profiles for the characterization of active tuberculosis, latent infection and end of treatment.JPEG by Sergio Díaz-Fernández (13136730)

“…Data also points to a significant change in the expression of CD38 (ESAT-6/CFP-10-specific) and Ki-67 (PPD- and ESAT-6/CFP-10-specific) after ending the anti-TB treatment regimen. …”

Table_1_Synergistic Antibacterial Activity of Designed Trp-Containing Antibacterial Peptides in Combination With Antibiotics Against Multidrug-Resistant Staphylococcus epidermidis.... by Dejing Shang (8037545)

“…In addition, the antibiotics in combination with the peptide significantly inhibited biofilm formation by MRSE 1208. A mechanistic study demonstrated that the Trp-containing peptides, especially I1WL5W and I4WL5W, which contain two tryptophan residues, disrupted bacterial inner and outer membranes, which promoted antibiotic delivery into the cytoplasm and access to cytoplasmic targets; however, L11W and L12W may have increased intracellular antibiotic concentrations by decreasing blaZ, tet(m) and msrA expression. …”

Image_2_Synergistic Antibacterial Activity of Designed Trp-Containing Antibacterial Peptides in Combination With Antibiotics Against Multidrug-Resistant Staphylococcus epidermidis.... by Dejing Shang (8037545)

“…In addition, the antibiotics in combination with the peptide significantly inhibited biofilm formation by MRSE 1208. A mechanistic study demonstrated that the Trp-containing peptides, especially I1WL5W and I4WL5W, which contain two tryptophan residues, disrupted bacterial inner and outer membranes, which promoted antibiotic delivery into the cytoplasm and access to cytoplasmic targets; however, L11W and L12W may have increased intracellular antibiotic concentrations by decreasing blaZ, tet(m) and msrA expression. …”

Image_4_Synergistic Antibacterial Activity of Designed Trp-Containing Antibacterial Peptides in Combination With Antibiotics Against Multidrug-Resistant Staphylococcus epidermidis.... by Dejing Shang (8037545)

“…In addition, the antibiotics in combination with the peptide significantly inhibited biofilm formation by MRSE 1208. A mechanistic study demonstrated that the Trp-containing peptides, especially I1WL5W and I4WL5W, which contain two tryptophan residues, disrupted bacterial inner and outer membranes, which promoted antibiotic delivery into the cytoplasm and access to cytoplasmic targets; however, L11W and L12W may have increased intracellular antibiotic concentrations by decreasing blaZ, tet(m) and msrA expression. …”

Image_3_Synergistic Antibacterial Activity of Designed Trp-Containing Antibacterial Peptides in Combination With Antibiotics Against Multidrug-Resistant Staphylococcus epidermidis.... by Dejing Shang (8037545)

“…In addition, the antibiotics in combination with the peptide significantly inhibited biofilm formation by MRSE 1208. A mechanistic study demonstrated that the Trp-containing peptides, especially I1WL5W and I4WL5W, which contain two tryptophan residues, disrupted bacterial inner and outer membranes, which promoted antibiotic delivery into the cytoplasm and access to cytoplasmic targets; however, L11W and L12W may have increased intracellular antibiotic concentrations by decreasing blaZ, tet(m) and msrA expression. …”

Image_1_Synergistic Antibacterial Activity of Designed Trp-Containing Antibacterial Peptides in Combination With Antibiotics Against Multidrug-Resistant Staphylococcus epidermidis.... by Dejing Shang (8037545)

“…In addition, the antibiotics in combination with the peptide significantly inhibited biofilm formation by MRSE 1208. A mechanistic study demonstrated that the Trp-containing peptides, especially I1WL5W and I4WL5W, which contain two tryptophan residues, disrupted bacterial inner and outer membranes, which promoted antibiotic delivery into the cytoplasm and access to cytoplasmic targets; however, L11W and L12W may have increased intracellular antibiotic concentrations by decreasing blaZ, tet(m) and msrA expression. …”

Behavior analysis in wild type and CHOP−/− mice with or without tunicamycin treatment. by Chang-Mu Chen (315897)

“…<p>(A). Passive avoidance was performed 1 day before and 5 days after administration of tunicamycin (0.1 mg/ml) in wild type and CHOP−/− mice. …”

PTPRM, link to tumour grade, nodal status, TNM staging and clinical outcomes of breast cancer. by Ping-Hui Sun (118782)

“…<p>A, PTPRM transcripts were decreased in the moderately and poorly differentiated cancer cells in comparison with well-differentiated tumour cells. …”

The acinar cells in the lacrimal gland. by Yunmeng Da (707943)

“…Compared with the SHAM group (Fig. 5, A), the endoplasmic reticulum was diluted and arranged in a disorderly fashion in the OVX groups. …”

Bifurcation Structure of Atypical and Conventional Models. by Christopher J. Knowlton (11450436)

“…Dynamics progress from a stable fixed point to a saddle node on an invariant limit cycle (SNIC) at -8 pA producing type 1 excitability. …”

Figure 3 from Synergistic Effects of PARP Inhibition and Cholesterol Biosynthesis Pathway Modulation by Anna Rutkowska (19676837)

“…<i>P</i> < 0.05, |log₂ fc| > log₂ (1.5)] modulated ones are indicated with name. Niraparib decreases CYP51A1 (lanosterol 14α-demethylase) enzyme levels (Supplementary Fig. …”

Neuronal Reverberation Is Strongest during SW Sleep by Sidarta Ribeiro (63675)

“…<div><p>(A) Rat 5 (CX) dramatically illustrates the state dependency of neuronal ensemble correlations, which are strongly increased by SW sleep but readily decreased by WK. …”

Higher Tregs frequency linked with PTB in individuals co-infected with or without HIV-1. by Jaideep S. Toor (627039)

“…<p><b>A.</b> Cumulative data depicting frequency of CD25⁺Foxp3⁺ Tregs within CD4⁺ T-cell in healthy controls (HC), patients with Pulmonary tuberculosis (PTB) and HIV-1 infected individuals with or without PTB co-infection. …”

Function of Calcium Ions in Cell Migration in Response to an EF. by Liang Guo (95092)

“…(B) The concentration of calcium in the anode culture media decreased in the larger chamber over time. (C) Migration speed in the vector direction decreased with time. * (p < 0.05) and ** (p < 0.01) vs EF 5 V/cm control. …”

Image_3_Bacterial RNA Contributes to the Down-Modulation of MHC-II Expression on Monocytes/Macrophages Diminishing CD4+ T Cell Responses.TIF by M. Ayelén Milillo (4325953)