Image_5_Abscisic acid-polyacrylamide (ABA-PAM) treatment enhances forage grass growth and soil microbial diversity under drought stress.JPEG by Xue Tang (4250896)

Table_5_Abscisic acid-polyacrylamide (ABA-PAM) treatment enhances forage grass growth and soil microbial diversity under drought stress.xlsx by Xue Tang (4250896)

Shape Stability of Octahedral PtNi Nanocatalysts for Electrochemical Oxygen Reduction Reaction Studied by <i>in situ</i> Transmission Electron Microscopy by Martin Gocyla (1486810)

“…Here, we perform <i>in situ</i> heating of compositionally segregated PtNi_1.5 octahedral nanoparticles inside a transmission electron microscope, in order to study their compositional and morphological changes. …”

Shape Stability of Octahedral PtNi Nanocatalysts for Electrochemical Oxygen Reduction Reaction Studied by <i>in situ</i> Transmission Electron Microscopy by Martin Gocyla (1486810)

“…Here, we perform <i>in situ</i> heating of compositionally segregated PtNi_1.5 octahedral nanoparticles inside a transmission electron microscope, in order to study their compositional and morphological changes. …”

A Protease Inhibitor as a Potential Therapy for Inherited Retinal Degeneration by Pratik Shah (281059)

Protein Quality Control Disruption by PKCβII in Heart Failure; Rescue by the Selective PKCβII Inhibitor, βIIV5-3 by Julio C. B. Ferreira (145311)

“…Importantly, inhibition of PKCβII, using a selective PKCβII peptide inhibitor (βIIV5-3), improved proteasomal activity and conferred protection in cultured neonatal cardiomyocytes. …”

Data_Sheet_1_Map2k5-Deficient Mice Manifest Phenotypes and Pathological Changes of Dopamine Deficiency in the Central Nervous System.ZIP by Yumeng Huang (591166)

“…Furthermore, Map2k5 mutant mice showed a decreased dopaminergic cell survival and tyrosine hydroxylase levels in nigrostriatal pathway, indicating a crucial role of MAP2K5 in regulating dopamine system in the central nervous system. …”

Image_3_Phosphodiesterase Type 5 Inhibitors Synergize Vincristine in Killing Castration-Resistant Prostate Cancer Through Amplifying Mitotic Arrest Signaling.TIF by Jui-Ling Hsu (515080)

“…Sildenafil, a specific phosphodiesterase type 5 (PDE5) inhibitor, has been suggested to display both cardioprotective and neuroprotective activities that provide a rationale for the combination with vincristine on the treatment against castration-resistant prostate cancer (CRPC). …”

Image_2_Phosphodiesterase Type 5 Inhibitors Synergize Vincristine in Killing Castration-Resistant Prostate Cancer Through Amplifying Mitotic Arrest Signaling.TIF by Jui-Ling Hsu (515080)

“…Sildenafil, a specific phosphodiesterase type 5 (PDE5) inhibitor, has been suggested to display both cardioprotective and neuroprotective activities that provide a rationale for the combination with vincristine on the treatment against castration-resistant prostate cancer (CRPC). …”

Image_4_Phosphodiesterase Type 5 Inhibitors Synergize Vincristine in Killing Castration-Resistant Prostate Cancer Through Amplifying Mitotic Arrest Signaling.TIF by Jui-Ling Hsu (515080)

“…Sildenafil, a specific phosphodiesterase type 5 (PDE5) inhibitor, has been suggested to display both cardioprotective and neuroprotective activities that provide a rationale for the combination with vincristine on the treatment against castration-resistant prostate cancer (CRPC). …”

Image_1_Phosphodiesterase Type 5 Inhibitors Synergize Vincristine in Killing Castration-Resistant Prostate Cancer Through Amplifying Mitotic Arrest Signaling.TIF by Jui-Ling Hsu (515080)

“…Sildenafil, a specific phosphodiesterase type 5 (PDE5) inhibitor, has been suggested to display both cardioprotective and neuroprotective activities that provide a rationale for the combination with vincristine on the treatment against castration-resistant prostate cancer (CRPC). …”

Image_7_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_2_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_10_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_6_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_4_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_1_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_9_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Image_3_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.tif by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”

Table_1_ARID5B regulates fatty acid metabolism and proliferation at the Pre-B cell stage during B cell development.pdf by Jaya Prakash Chalise (16506906)

“…Dysregulation of this process may result in the failure to produce functional B cells and pose a risk of leukemic transformation. Here, we report that AT rich interacting domain 5B (ARID5B), a B cell acute lymphoblastic leukemia (B-ALL) risk gene, regulates B cell development at the Pre-B stage. …”