Table1_Mitral valve aneurysms: echocardiographic characteristics, formation mechanisms, and patient outcomes.xlsx by Yi Wang (32470)

“…</p>Conclusions<p>We recommend using echocardiography as a highly sensitive method for MVA diagnosis. …”

Fetal and Childhood Exposure to Phthalate Diesters and Cognitive Function in Children Up to 12 Years of Age: Taiwanese Maternal and Infant Cohort Study by Han-Bin Huang (322180)

“…A total of 110, 79, 76, and 73 children were followed up at ages 2, 5, 8, and 11, respectively. …”

Cellular starvation in <i>C. trachomatis</i>-infected cells. by Márta Szaszák (214158)

“…Quantitative analysis of τ₂-NAD(P)H (n = 54 ROIs from three independent experiments; mean±SEM). Detailed results of statistical analysis are shown in <a href="http://www.plospathogens.org/article/info:doi/10.1371/journal.ppat.1002108#ppat.1002108.s012" target="_blank">Table S5A</a>. …”

Supplementary Materials - Antitumor activity of new chemical compounds in triple negative mammary adenocarcinoma models by Maria V Giolito (8499600)

“…According to the need for the development of new anticancer agents, we have synthetized novel bioactive compounds and aimed to determine their antitumor action. …”

Interactions between TSC2, TBC1D7 and the TSC1delex proteins. by Arthur Jorge Santiago Lima (549061)

“…TSC1 delex18 and delex22 showed a significant decrease in the amount of coimmunoprecipitated TBC1D7 (unpaired t-test p values<0.05; indicated with asterisks). …”

The effect of Pam3CSK4 on adhesion of HMCLs to BMSCs. by Jahangir Abdi (400453)

“…Pam3CSK4 decreased adhesion of OPM-1, OPM-2, and NCI-H929 cell lines to HS-5 in a dose-dependent manner. …”

DOR binding is enhanced by CB₁R ligands in membranes of cells expressing CB₁R and DOR. by Ittai Bushlin (191060)

“…Data represent Mean ± SEM (n = 3 experiments in triplicate). <b>B,</b> Membranes from N2A-DOR cells in which CB₁R expression was decreased by siRNA transfection were treated with 100 fM – 10 µM Hu-210 in the presence of 0.5 nM [³H]DPDPE. …”

Data Sheet 1_Investigating the molecular mechanisms associated with ulcerative colitis through the application of single-cell combined spatial transcriptome sequencing.docx by Hua Huang (190195)

“…Immunohistochemical analysis of UC lesion samples revealed diminished expression levels of GNG5 and elevated expression levels of TIMP1. A dextran sulfate sodium (DSS)-induced colitis mouse model was developed, demonstrating that the protein expression levels of GNG5 in the colonic tissue of model mice were significantly decreased compared to controls w)ile the expression levels of TIMP1 were increased (p < 0.01). …”

The NK1-receptor antagonist ezlopitant significantly inhibits both sucrose and ethanol operant self-administration in rats. by Pia Steensland (362091)

“…<p>Ezlopitant (2, 5, 10 mg/kg i.p.) dose dependently decreases self-administration of 5% sucrose (<b>A</b>). …”

Clinical consequences of weight loss during stem cell transplantation. by Christina T. Rieger (838986)

“…<p>A: No difference in overall survival between patients that lose ≥ 5% of BMI during SCT B: Similar result for a BMI decrease of ≥ 10%. …”

Comparison of diet, age, and insulin signaling effects on CHCs. by Tatyana Y. Fedina (114609)

“…<p>Arrows indicate an increase or decrease in the relative abundance of individual CHCs: 1) for aging - from young to old age on balanced diets <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0049799#pone.0049799-Kuo1" target="_blank">[18]</a>, 2) for protein – based on change from S5Y5 to S5Y20, and for sugar – based on change from S5Y5 to S20Y5, 3) for insulin signaling (IIS) - change in transgenic flies with increased insulin signaling (via insulin receptor overexpression - InR^OX) or decreased insulin signaling (insulin substrate mutants <i>chico</i>) compared to controls <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0049799#pone.0049799-Kuo2" target="_blank">[19]</a>. …”

Movie S1 - Quantifying Exocytosis by Combination of Membrane Capacitance Measurements and Total Internal Reflection Fluorescence Microscopy in Chromaffin Cells by Ute Becherer (74949)

“…This cell was stimulated for 2 s by depolarizing pulses and is shown 4 s prior to and 8.5 s after stimulation. Secretion of vesicles seen as a sharp decrease of fluorescence of individual spots (highlighted by white arrows) occurs simultaneously to the increase in membrane capacitance and only during the stimulation period (marked in red). …”

Modulatory effects of dopamine shown as changes in state space (interpreted after 30)(see also text). by Nico J. M. van Beveren (51667)

“…<p><a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0002577#pone-0002577-g005" target="_blank">Figure 5</a>, top: increasing levels of dopamine augment the signal-to-noise ratio: the basin of the attractor associated with the most salient signal increases while the basins of attraction of the other attractors decrease. …”

Image_2_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Image_7_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Image_4_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Image_6_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Image_8_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Image_9_The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation.tif by Wenhui Zhuang (10007378)

“…Kv7.4 channels co-expressed with wild-type (WT) CaM show inhibited activation when intracellular calcium levels increase, while Kv7.4 channels co-expressed with CaM₁₂₃₄ or CaM₃ are insensitive to calcium. Mutations C156A, C157A, C158V, R159, and R161A, which are located within the Kv7.4 S2–S3 loop, dramatically facilitate activation of Kv7.4 channels co-expressed with WT CaM but have no effect on activation of Kv7.4 channels co-expressed with CaM₃, indicating that these five mutations decrease the inhibitory effect of Ca²⁺/CaM. …”

Data_Sheet_1_4DEMON: Integrating 40 Years of Data on PCB and Metal Contamination in Marine Sediments of the Belgian Part of the North Sea.docx by Hong Minh Le (10887786)

“…Trend analysis revealed a strong decrease in PCB concentrations in the nineteen eighties and nineties, followed by a slight increase over the last decade. …”