Showing 17,801 - 17,820 results of 103,202 for search '(( 5 ((ng decrease) OR (a decrease)) ) OR ( 50 ((nn decrease) OR (we decrease)) ))', query time: 1.55s Refine Results
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    Deficiency of Calcium-Independent Phospholipase A2 Beta Induces Brain Iron Accumulation through Upregulation of Divalent Metal Transporter 1 by Goichi Beck (818821)

    Published 2015
    “…The present study aimed to clarify the mechanism underlying brain iron accumulation during the deficiency of calcium-independent phospholipase A2 beta (iPLA<sub>2</sub>β), which is encoded by the <i>PLA2G6</i> gene. …”
  11. 17811

    Mice deficient in NKLAM have attenuated inflammatory cytokine production in a Sendai virus pneumonia model by Donald W. Lawrence (4923328)

    Published 2019
    “…Here, we present novel data describing a role for NKLAM in regulating the immune response to Sendai virus (SeV), a murine model of paramyxoviral pneumonia. …”
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    <i>Drosophila</i> Spidey/Kar Regulates Oenocyte Growth via PI3-Kinase Signaling by Einat Cinnamon (2922108)

    Published 2016
    “…Several enzymes of fatty acid, triglyceride and hydrocarbon metabolism are required in oenocytes primarily for lipid droplet induction rather than for cell growth. In contrast, a very long chain fatty-acyl-CoA reductase (FarO) and a putative lipid dehydrogenase/reductase (Spidey, also known as Kar) not only promote lipid droplet induction but also inhibit oenocyte growth. …”
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    Passenger mutations accurately classify human tumors by Marina Salvadores (6589187)

    Published 2019
    “…The advantage of passenger mutations was upheld under high rates of false negative mutation calls and with exome sequencing, even though overall accuracy decreased. We suggest whole genome sequencing is valuable for classifying tumors because it captures global patterns emanating from mutational processes, which are informative of the underlying tumor biology.…”
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    Antiarrhythmic Hit to Lead Refinement in a Dish Using Patient-Derived iPSC Cardiomyocytes by John R. Cashman (354526)

    Published 2021
    “…Long QT syndrome type-3 (LQT3) is a congenital form of VA caused by cardiac sodium channel (I<sub>NaL</sub>) SCN5A mutations that prolongs cardiac action potential (AP) and enhances I<sub>NaL</sub> current. …”