Showing 9,181 - 9,200 results of 45,247 for search '(( 5 ((ng decrease) OR (mean decrease)) ) OR ( 50 ((teer decrease) OR (a decrease)) ))', query time: 1.02s Refine Results
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    Population-level vaccine effectiveness (<i>VE</i>), assuming that density dependence acts on the fecundity of established parasites, and that 50% of the host population is effectiv... by Lorenzo Sabatelli (248918)

    Published 2010
    “…<p>Vaccines can reduce the host susceptibility to infection (A–B), by a proportion <i>VS</i>, or the female parasite reproductive rate (C–D), by a proportion <i>VR</i>, or the parasite lifespan (E–F), by a proportion <i>VM</i>. …”
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    LPC-DHA affects membrane phospholipid saturation. by Jia Pei Chan (5593388)

    Published 2018
    “…<p>Targeted lipidomic analysis of NSC<sup>WT</sup> and NSC<sup>KO</sup> cells treated with or without 50 μM LPC-DHA for 16 hours. (A) NSC<sup>WT</sup> cells showed increased DHA in PC, PE, and PS relative to NSC<sup>KO</sup> cells. …”
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    Glycochenodexycholic (GCDCA) causes lumen obstruction and subepithelial fibrosis of neonatal extrahepatic bile ducts (EHBDs), while ursodeoxycholic acid (UDCA) attenuates GCDCA tox... by Miri Dotan (12250909)

    Published 2022
    “…<p>(a) Neonatal EHBD were dissected and incubated for 24 hours in biliary epithelial cell media, with or without GCDCA 5mM, and with GCDCA 5mM combined with UDCA acid 5mM. …”
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    Data_Sheet_1_Herpes Simplex Virus 1 UL2 Inhibits the TNF-α–Mediated NF-κB Activity by Interacting With p65/p50.pdf by Mingsheng Cai (286548)

    Published 2020
    “…<p>Herpes simplex virus 1 (HSV-1) is a large double-stranded DNA virus that encodes at least 80 viral proteins, many of which are involved in the virus–host interaction and are beneficial to the viral survival and reproduction. …”
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    Csf1r or Mer inhibition delays liver regeneration via suppression of Kupffer cells by Juan A. Santamaria-Barria (6655667)

    Published 2019
    “…At 36h after PH, Csf1r inhibition increased serum ALT and histological liver injury, and decreased liver cell proliferation. A small molecule inhibitor of Mer did not alter the percentage of KCs or their proliferation and just modestly delayed LR. …”
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