Effect of obesity on contraction of mesenteric arteries to angiotensin II and norepinephrine. by Graziela N. Hagihara (623403)

Contribution of angiotensin II receptors activation to the vascular effects of angiotensin. by Graziela N. Hagihara (623403)

Effect of angiotensin II on eNOS phosphorylation in mesenteric arteries. by Graziela N. Hagihara (623403)

PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

“…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”

miR-27a protects human mitral valve interstitial cell from TNF-α-induced inflammatory injury via up-regulation of NELL-1 by Honglei Chen (125181)

Expression of CRYAB in normal and tumor tissue samples across different cancer types. by Rasool Saghaleyni (10526301)

DNMT1 reduces cisplatin sensitivity partially through downregulating FOXO3a in ovarian cancer cells by Chong Guo (5819105)

“…Knocking down of DNMT1 could decrease the IC₅₀ of cisplatin. Treatment with cisplatin may reduce FOXO3a expression in OC cells. …”

The number of days until tumor confirmation is inversely proportional to the amount of inoculated F98 cells in the five rat groups. by Velislava Zoteva (17720500)

Flow cytometer analysis of the different cell populations in the absence or presence of 1 μg/ml doxycycline for 5 days. by Nicolas Jullien (82773)

Hopping into a hot seat: Role of DNA structural features on IS<i>5</i>-mediated gene activation and inactivation under stress - Fig 7 by M. Zafri Humayun (4217524)

“…The G(x) values are decreased for the <i>ORF-3</i> mutation (blue), meaning that the duplex is further destabilized relative to the wild type sequence. …”

Effects of angiotensin II on ERK 1/2 phosphorylation in mesenteric arteries. by Graziela N. Hagihara (623403)

Efficiency of explaining and predicting risk premium in different samples. by Mihály Ormos (697939)

<i>Shigella</i> infection increases PML-NB number. by Saima M. Sidik (590022)

Validation of hTERT mRNA as a direct target of miR-512-5p in CNE cells. by Jun Li (6494)

“…(B) MiR-512-5p resulted in a significant decrease in luciferase expression in hTERT-3’UTR transfected cells compared with the scramble, while MiR-512-5p caused no statistical change in luciferase expression in mut-hTERT-3’UTR compared with the scramble(C) miR-512-5p mimic led to down-regulation of hTERT and TRF2 in CNE cells. …”

Suction-electrode recordings from WT and <i>TrUx;GapOx</i> rods. by Ala Morshedian (4165957)

Simulation fitting COVID-19 data from Chile. by Fernando Córdova-Lepe (9727230)