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ng decrease » _ decrease (Expand Search), we decrease (Expand Search), gy decreased (Expand Search)
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
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Showing 2,001 - 2,020 results of 21,278 for search '(( 5 ((ng decrease) OR (nn decrease)) ) OR ( 100 ((mean decrease) OR (a decrease)) ))', query time: 1.04s Refine Results
  1. 2001
    Purified recombinant WT-C100Flag dimer and trimer showed reduced Aβ production.

    Purified recombinant WT-C100Flag dimer and trimer showed reduced Aβ production. by Joo In Jung (650746)

    Published 2014
    “…<p>(A) The purified recombinant WT-C100Flag monomer, dimer, and trimer eluates were loaded on a SDS-PAGE gel. …”
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  2. 2002
    GluN2A(D731N) reduces the agonist potency.

    GluN2A(D731N) reduces the agonist potency. by Kai Gao (120150)

    Published 2017
    “…(<b>C,E</b>) The composite glutamate (in the presence of 100 μM glycine) concentration-response curves reveal a significant decrease in glutamate potency in both di-heteromeric (<b>C</b>) and tri-heteromeric (<b>E</b>) GluN2A(D731N)-containing NMDARs compared to wild type receptors. …”
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  3. 2003
    Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx

    Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  4. 2004
    Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF

    Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  5. 2005
    Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx

    Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  6. 2006
    Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF

    Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  7. 2007
    Image_3_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF

    Image_3_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  8. 2008
    Image_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF

    Image_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

    Published 2020
    “…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”
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  9. 2009
    Data_Sheet_1_Molecular Mechanisms by Which S100A4 Regulates the Migration and Invasion of PGCCs With Their Daughter Cells in Human Colorectal Cancer.zip

    Data_Sheet_1_Molecular Mechanisms by Which S100A4 Regulates the Migration and Invasion of PGCCs With Their Daughter Cells in Human Colorectal Cancer.zip by Fei Fei (794241)

    Published 2020
    “…The tumorigenic and metastatic ability of PGCCs with their daughter cells in vivo was significantly stronger compared to the untreated cells, which was significantly decreased after S100A4 knockdown. Moreover, the expression of S100A4-related proteins was positively correlated with the malignancy degree of human CRC, and maintained a high level in lymph node metastasis. …”
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  10. 2010
    GluN2A(D731N) enhances sensitivity to endogenous proton and zinc ions.

    GluN2A(D731N) enhances sensitivity to endogenous proton and zinc ions. by Kai Gao (120150)

    Published 2017
    “…Di-heteromeric (h2A-D731N), one-copy and two-copy mutant tri-heteromeric (D731N/2A and D731N/D731N) receptors show a decreased current ratio, indicating enhanced proton sensitivity. …”
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  11. 2011
    ConnectivityDiagram_NN2SF_p10.png from Interaction network effects on position- and velocity-based models of collective motion

    ConnectivityDiagram_NN2SF_p10.png from Interaction network effects on position- and velocity-based models of collective motion by Ali Emre Turgut (780622)

    Published 2020
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  12. 2012
    ConnectivityDiagram_NN2ER_p10.png from Interaction network effects on position- and velocity-based models of collective motion

    ConnectivityDiagram_NN2ER_p10.png from Interaction network effects on position- and velocity-based models of collective motion by Ali Emre Turgut (780622)

    Published 2020
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  13. 2013
    ConnectivityDiagram_NN2SF_p02.png from Interaction network effects on position- and velocity-based models of collective motion

    ConnectivityDiagram_NN2SF_p02.png from Interaction network effects on position- and velocity-based models of collective motion by Ali Emre Turgut (780622)

    Published 2020
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  14. 2014
    ConnectivityDiagram_NN2ER_p02.png from Interaction network effects on position- and velocity-based models of collective motion

    ConnectivityDiagram_NN2ER_p02.png from Interaction network effects on position- and velocity-based models of collective motion by Ali Emre Turgut (780622)

    Published 2020
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  15. 2015
    sAxl levels in LN patients and healthy subjects.

    sAxl levels in LN patients and healthy subjects. by Ioannis Parodis (2851211)

    Published 2019
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  16. 2016
    Conditional ablation of <i>Ybx1</i> leads to a reduction in cortical progenitor cell pool and a decrease in cortical thickness.

    Conditional ablation of <i>Ybx1</i> leads to a reduction in cortical progenitor cell pool and a decrease in cortical thickness. by Jian Zhang (1682)

    Published 2025
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  17. 2017
    Effect of high-fructose (HFr) supplementation (500 μM) on markers of oxidative stress and HO-1 and SIRT1 gene expressions in hepatocytes treated with and without CoPP (5 μM) and SnMP (5 μM).

    Effect of high-fructose (HFr) supplementation (500 μM) on markers of oxidative stress and HO-1 and SIRT1 gene expressions in hepatocytes treated with and without CoPP (5 μM) and Sn... by Komal Sodhi (540891)

    Published 2015
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  18. 2018
    Expression of wild-type KCNQ2 but not KCNQ2-A343D or KCNQ2-R353G decreases neuronal excitability.

    Expression of wild-type KCNQ2 but not KCNQ2-A343D or KCNQ2-R353G decreases neuronal excitability. by John P. Cavaretta (606562)

    Published 2014
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  19. 2019
    Concentrations of biomarkers—intestinal fatty acid-binding protein (IFABP), syndecan-1, lipopolysaccharide-binding protein (LBP), soluble CD14 (sCD14) and LBP:sCD14 ratio—over 24 hours with a low fat, low carbohydrate meal plan after the placebo (saline) injection.

    Concentrations of biomarkers—intestinal fatty acid-binding protein (IFABP), syndecan-1, lipopolysaccharide-binding protein (LBP), soluble CD14 (sCD14) and LBP:sCD14 ratio—over 24 h... by Tamara Mogilevski (14239682)

    Published 2024
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  20. 2020
    Cell culture and amastigote assay workflow- THP 1 monocyte were cultured and passaged every alternate day for drug testing experiments: Day 1–500 μl of THP 1 PMA treated cells were inoculated in chamber slide for 24 hours at 37°C for differentiation in macrophages; Day 2- <i>Lmajor</i> metacyclic promastigotes were infected into these macrophages and incubated for phagocytoses at a ratio of 1:10 at 32°C for 2 hours.

    Cell culture and amastigote assay workflow- THP 1 monocyte were cultured and passaged every alternate day for drug testing experiments: Day 1–500 μl of THP 1 PMA treated cells were... by Harshita Sharma (4836180)

    Published 2023
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