Transcription factor binding site analysis of HITS upregulated by different IBD-ORFs by gProfiler. by Mohamad Karaky (8910497)

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Shared impact on transcriptome for the ORFs PTGIR, NFKB1, SLC39A11 and ZBTB40. by Mohamad Karaky (8910497)

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Impact of LPS on PTGIR expression in THP-1. by Mohamad Karaky (8910497)

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Forward and reverse primer sequences 5´-3´ used in the study. by Mohamad Karaky (8910497)

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Numeric data for the graphs in the main and the supporting figures. by Mohamad Karaky (8910497)

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Shared impact on transcriptome for the ORFs ATG16L1, IFIH1 and IRF5. by Mohamad Karaky (8910497)

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Shared impact on transcriptome for the ORFs SLC39A11 and NOD2. by Mohamad Karaky (8910497)

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S5 Table - by Mohamad Karaky (8910497)

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The impact of different concentrations of PTGIR agonist on CP genes. by Mohamad Karaky (8910497)

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Gene expression of PTGIR and PTGER4 in hiPSC monocyte/macrophages derived. by Mohamad Karaky (8910497)

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Impact of IBD gene candidate ORFs on the THP-1 transcriptome. by Mohamad Karaky (8910497)

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The impact of PTGIR agonist on CP genes at different time. by Mohamad Karaky (8910497)

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Germination percent response of <i>I</i>. <i>rugosum</i> as affected by decreasing osmotic potential (MPa) plotted over time (in days after sowing, DAS), modeled with the use of eq... by Charlemagne Alexander A. Lim (795608)

The synergistic effect of HDAC and proteasome inhibition is consistent within each drug class. by Aimée N. Laporte (3617987)

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HDAC inhibition prevents aggresome formation in response to proteasome inhibitors, and combination treatment leads to endoplasmic reticulum stress. by Aimée N. Laporte (3617987)

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High-throughput drug screen reveals HDAC and proteasome inhibitors as potent drug classes against synovial sarcoma. by Aimée N. Laporte (3617987)

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HDAC and proteasome inhibition leads to apoptosis via pro-apoptosis protein activation, ROS production and caspase activation. by Aimée N. Laporte (3617987)

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Overexpression of wild-type and mutant ALDH3A1 retards hTCEpi cell proliferation. by Vindhya Koppaka (845151)

“…After 4 d, cells were trypsinized and viable cells were counted using trypan blue exclusion on a hemocytometer. Consecutively, 2.0 x 10⁵ cells were transferred to a new dish and this process repeated for a total of 8 passages, i.e., for 32 d until treated cells showed decreasing cell numbers. …”

The top 100 TCs with highest RR for all-cause, CVDs, and RDs mortality. by Wenzhong Huang (538470)

Effect of 1N11 on cultured endothelial cell migration impairment by 3F8 or aPL, and on aPL blunting of carotid artery reendothelialization in vivo. by Chieko Mineo (725831)