Showing 6,221 - 6,240 results of 28,667 for search '(( 5 ((ng decrease) OR (nn decrease)) ) OR ( 50 ((we decrease) OR (a decrease)) ))', query time: 0.74s Refine Results
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    PCAIs inhibit NCI-H23 cell line viability. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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    PCAIs disrupt actin filaments in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  4. 6224

    PCAIs suppress 3D NCI-H23 cell invasion. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  5. 6225

    PCAIs suppress NCI-H23 cancer cell migration. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
  6. 6226

    PCAIs induce apoptosis in NCI-H23 cells. by Matthew D. Gregory (19929096)

    Published 2024
    “…Polyisoprenylated Cysteinyl amide Inhibitors (PCAIs) constitute a group of potential cancer therapy agents that we designed to specifically disrupt and suppress hyperactive G-protein signaling, such as that caused by mutated RAS proteins. …”
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    Image_5_Experimental Myocardial Infarction Elicits Time-Dependent Patterns of Vascular Hypoxia in Peripheral Organs and in the Brain.TIF by Hélène David (8402883)

    Published 2021
    “…Acute LV dysfunction was associated with global hypoxia, specifically a decrease in sO<sub>2</sub> level in the brain (−5.9%), kidney (−6.4%), and liver (−7.3%) at 4 and 24 h post-MI. …”
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    DNMT1 reduces cisplatin sensitivity partially through downregulating FOXO3a in ovarian cancer cells by Chong Guo (5819105)

    Published 2025
    “…Knocking down of DNMT1 could decrease the IC<sub>50</sub> of cisplatin. Treatment with cisplatin may reduce FOXO3a expression in OC cells. …”
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    Suppression by extracellular QRNA from TNP Ts Sup free of exosomes, or miR-150 alone, associating with B-cell derived Ag-specific exosomes from the assayed CS-effector cell mixture... by Krzysztof Bryniarski (731989)

    Published 2015
    “…</b></u> Similarly, two day immune B-1 B cell-derived exosomes, supplemented with decreasing doses of miR-150 alone, mediated suppression of TNP-CS-effector cell adoptive transfer (Groups E-G), down to a dose of 750pg per eventual recipient, which is 50 femtomoles per eventual recipient (Group G). …”
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