Showing 441 - 460 results of 38,396 for search '(( 5 ((ng decrease) OR (we decrease)) ) OR ( 100 ((nn decrease) OR (a decrease)) ))', query time: 0.63s Refine Results
  1. 441

    Image 3_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.tif by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  2. 442

    Data Sheet 1_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.csv by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  3. 443

    Image 1_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.tif by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  4. 444

    Image 2_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.tif by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  5. 445

    Data Sheet 4_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.csv by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  6. 446

    Data Sheet 3_Liver X receptor agonist T0901317 alleviates sepsis-induced acute lung injury by enhancing macrophage autophagy.csv by Ben Wang (250897)

    Published 2025
    “…Following S100A8 knockdown in the macrophages with S100A8-siRNA, the IL-6 expression was noted to decrease in cells treated with T0+LPS+S100A8-siRNA than those treated with LPS+T0. …”
  7. 447

    DataSheet1_Blocking S100A9-signaling is detrimental to the initiation of anti-tumor immunity.pdf by Melike Fusun Demir (19919784)

    Published 2024
    “…This effect was caused by a reduction in the tumor immune infiltration to about half of untreated controls, and the reduction was made up of a 5-fold decrease in Ly6C<sup>high</sup> monocytic cells. …”
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  16. 456

    Data Sheet 2_S100A4 targets PPP1CA/IL-17 to inhibit the senescence of sheep endometrial epithelial cells.pdf by Xiyao Jiao (18695935)

    Published 2024
    “…</p>Methods<p>The protein PPP1CA, which interacts with S100A4, was detected by immunoprecipitation-mass spectrometry of overexpression and knockdown of S100A4 and PPP1CA. …”
  17. 457

    Data Sheet 3_S100A4 targets PPP1CA/IL-17 to inhibit the senescence of sheep endometrial epithelial cells.pdf by Xiyao Jiao (18695935)

    Published 2024
    “…</p>Methods<p>The protein PPP1CA, which interacts with S100A4, was detected by immunoprecipitation-mass spectrometry of overexpression and knockdown of S100A4 and PPP1CA. …”
  18. 458

    Data Sheet 1_S100A4 targets PPP1CA/IL-17 to inhibit the senescence of sheep endometrial epithelial cells.pdf by Xiyao Jiao (18695935)

    Published 2024
    “…</p>Methods<p>The protein PPP1CA, which interacts with S100A4, was detected by immunoprecipitation-mass spectrometry of overexpression and knockdown of S100A4 and PPP1CA. …”
  19. 459
  20. 460

    Juvenile demyelination leads to a substantial decrease in potassium currents in PV interneurons of the PFC. by Sara Hijazi (21656615)

    Published 2025
    “…Scale: 500 pA, 100 ms. <b>B</b>. I–V curves showing a significant decrease in Kv amplitude in PV interneurons from mice that underwent juvenile demyelination. …”