Mitochondrial morphology assay. حسب Dong Ma (584680)

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SMC-specific knockout of Klf5 (smcKlf5^−/−) exacerbates the progression of Ang II–induced AAA by facilitating ROS formation. حسب Dong Ma (584680)

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The correlation of the mitochondrial dynamics–related genes with Klf5 in mouse VSMCs. حسب Dong Ma (584680)

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Numerical raw data. حسب Dong Ma (584680)

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The primers for real-time PCR. حسب Dong Ma (584680)

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ERK signaling and expression of β-arrestin2 in Ang II–induced mouse AAA models as well as in WT or Klf5^−/− VSMCs. حسب Dong Ma (584680)

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Demographic and clinical characteristics of patients with AAA. حسب Dong Ma (584680)

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The expression of eIF5a, Fis1, Pink1, Drp1, Mfn1, and Mtfr1 and the analysis of mitochondrial morphology. حسب Dong Ma (584680)

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The effect of ML264 (Klf5 inhibitor) on aneurysm formation induced by Ang II infusion in the early and late stages. حسب Dong Ma (584680)

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Cardiac function assessed by echocardiography in Ang II–infused young (3 months) or old (18 months) WT and smcKlf5^−/− mice. حسب Dong Ma (584680)

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Klf5 deficiency in mouse VSMCs leads to mitochondrial fission. حسب Dong Ma (584680)

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ATP assay. حسب Dong Ma (584680)

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Representative TUNEL- and DAPI-stained sections from the abdominal aortas of young and old WT and smcKlf5^−/− mice following 28 days of Ang II infusion. حسب Dong Ma (584680)

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eIF5a affects the expression of mitochondrial dynamics–related genes. حسب Dong Ma (584680)

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Young (3 months) or old (18 months) WT and smcKlf5^−/− mice were infused with Ang II for 28 days. حسب Dong Ma (584680)

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Klf5 down-regulation leads to human VSMC senescence. حسب Dong Ma (584680)

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CcnA mutation of Loop A decreases the interaction with the 5′ UTR of P2-<i>ctrA</i> mRNA and 5′ UTR of <i>gcrA</i> mRNA. حسب Wanassa Beroual (5314028)

Statistically significant union pathways refer to the miRNAs with a decreased expression on day 1. حسب Morgana Lüdtke Azevedo (15285409)

Decreased activity of FASII favors the cold growth of the <i>ΔcshA</i> strain. حسب Vanessa Khemici (814639)

"…<i>fapR</i> mRNA levels were quantified by RT-qPCR using 16S rRNA as reference gene on total RNAs extracted from <i>wt</i> strain (PR01), <i>ΔcshA</i> strain (PR01-ΔcshA), and suppressor strains <i>ΔcshA/accC</i>^<i>M385V</i> (C51), <i>ΔcshA/accC</i>^<i>T183I</i> (sup30), <i>ΔcshA/accD</i>^<i>F253V</i> (sup17), <i>ΔcshA/accD</i>^<i>A164V</i> (sup16), <i>ΔcshA/birA</i>^{<i>D320F-FsX28</i>} (C58), <i>ΔcshA/birA</i>^{<i>R280stop</i>} (sup1) and <i>ΔcshA/bioY</i>^{<i>P123R-FsX1</i>} (C66), all at exponential growth phase in MH medium at 25°C. n = 5 for <i>ΔcshA</i>, <i>4 for wt</i> and 3 for all others. …"

Seven out of the 11 human cytidine deaminases are more highly expressed in tumor versus normal tissue and their higher expression is correlated with a decrease in survival. حسب Mason A. McCool (11889842)