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point decrease » point increase (Expand Search)
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26601
Bidirectional Optical Control of Proton Motive Force in Escherichia coli Using Microbial Rhodopsins
Published 2024“…These alterations in PMF correspond to +146 mV (<i>Rm</i>XeR) and −140 mV (AR3), respectively. …”
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26602
Bidirectional Optical Control of Proton Motive Force in Escherichia coli Using Microbial Rhodopsins
Published 2024“…These alterations in PMF correspond to +146 mV (<i>Rm</i>XeR) and −140 mV (AR3), respectively. …”
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26603
Bidirectional Optical Control of Proton Motive Force in Escherichia coli Using Microbial Rhodopsins
Published 2024“…These alterations in PMF correspond to +146 mV (<i>Rm</i>XeR) and −140 mV (AR3), respectively. …”
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26604
Bidirectional Optical Control of Proton Motive Force in Escherichia coli Using Microbial Rhodopsins
Published 2024“…These alterations in PMF correspond to +146 mV (<i>Rm</i>XeR) and −140 mV (AR3), respectively. …”
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26605
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26606
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26607
The detailed risk of bias assessment.
Published 2025“…Therefore, the existence of a dose-response relationship can indicate a causal relationship between music therapy and the improvement of neonatal outcomes.…”
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26608
Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26609
Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26610
Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26611
Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26612
Image_3_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26613
Image_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF
Published 2020“…Interestingly, Rv1768 binding to S100A9 also disturbs the metabolism of arachidonic acid by activating 5-lipoxygenase, increasing lipotoxin A4, and down-regulating cyclooxygenase-2 and prostaglandin E2 expression, thus, promoting mycobacterial survival. …”
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26614
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26615
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26616
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26617
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26618
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26619
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26620