Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_4_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_6_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_1_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.pdf by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_2_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Data_Sheet_3_P16INK4a Deletion Ameliorates Damage of Intestinal Epithelial Barrier and Microbial Dysbiosis in a Stress-Induced Premature Senescence Model of Bmi-1 Deficiency.docx by Jiawen Zhou (2323381)

“…P16^INK4a deletion could maintain barrier function and microbiota balance in Bmi-1^–/– mice through strengthening formation of TJ and decreasing macrophages-secreted TNF-α induced by Desulfovibrio entering the intestinal epithelium. …”

Training and salivary data set. by Davide Curzi (466778)

“…Moreover, this study investigated athletes’ hormonal response to a typical weekly conditioning session (5 days before match: MD-5), at T1 and T2, to detect changes in players’ recovery capability over time. …”

Timeline of the study. by Davide Curzi (466778)

“…Moreover, this study investigated athletes’ hormonal response to a typical weekly conditioning session (5 days before match: MD-5), at T1 and T2, to detect changes in players’ recovery capability over time. …”

The constitutively active form of insulin receptor and knockdown of FoxO block high temperature-mediated body fat loss. by Tucker Hopkins (20790529)

Foxo3a-dependent miR-633 regulates chemotherapeutic sensitivity in gastric cancer by targeting Fas-associated death domain by Xin Pang (249322)

A Model For A Narrow-Cut Fractionating Column by Farag, Hassan A.

“…A mathematical model was developed to simulate the operation of a narrow-cut fractionating column. …”
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S1 File - by Chantelle M. de Vet (16535257)

β cell-specific deletion of HMG-CoA (3-hydroxy-3-methylglutaryl-coenzyme A) reductase causes overt diabetes due to reduction of β cell mass and impaired insulin secretion by Ada Admin (8524211)

Crossing of transcriptome and metabolome profiles of the arginine pathway, highlighting the enzymes with differential gene expression based on the RNA-seq data. by Juliana Ide Aoki (3109917)

“…<p>Representation of the arginine pathway and FPKM expression levels highlighting in gray boxes the enzymes with differential gene expression: (A) argininosuccinate synthase (EC6.3.4.5), (B) acetylornithine deacetylase (EC3.5.1.16), (C) glutamate 5-kinase (EC2.7.2.11), (D) spermidine synthase (EC2.5.1.16), (E) arginase (EC3.5.3.1), (F) pyrroline 5-carboxylase reductase (EC1.5.1.2) and (G) pyrroline 5-carbolylate dehydrogenase (EC1.2.1.88). …”

Serum Glycated Albumin to Guide the Diagnosis of Diabetes Mellitus by Wan-Chen Wu (845865)

“…When serum GA value of 14% and 17% were used to exclude and diagnose diabetes, respectively, the sensitivity improved to 83.3%, with a slightly decrease in specificity (98.2%), but a significant increase in OGTT% (35%). …”

Table_5_Spatial-temporal analysis of hepatitis E in Hainan Province, China (2013-2022): insights from four major hospitals.XLSX by Zhi Yun (2139964)

Geochemical and microbiological data obtained from downgradient well CD01 at the Rifle site, showing (A/B) the concurrent increase in Fe(II) and decrease in aqueous U(VI) associate... by Michael J. Wilkins (385629)

<i>S</i>. Typhimurium coordinates protein synthesis with protein folding capacity. by Carissa Chan (18310140)