The correlation of the mitochondrial dynamics–related genes with Klf5 in mouse VSMCs. by Dong Ma (584680)

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Numerical raw data. by Dong Ma (584680)

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The primers for real-time PCR. by Dong Ma (584680)

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ERK signaling and expression of β-arrestin2 in Ang II–induced mouse AAA models as well as in WT or Klf5^−/− VSMCs. by Dong Ma (584680)

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Demographic and clinical characteristics of patients with AAA. by Dong Ma (584680)

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The expression of eIF5a, Fis1, Pink1, Drp1, Mfn1, and Mtfr1 and the analysis of mitochondrial morphology. by Dong Ma (584680)

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The effect of ML264 (Klf5 inhibitor) on aneurysm formation induced by Ang II infusion in the early and late stages. by Dong Ma (584680)

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Cardiac function assessed by echocardiography in Ang II–infused young (3 months) or old (18 months) WT and smcKlf5^−/− mice. by Dong Ma (584680)

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Klf5 deficiency leads to mitochondrial dyfunction in VSMCs. by Dong Ma (584680)

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Klf5 deficiency in mouse VSMCs leads to mitochondrial fission. by Dong Ma (584680)

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ATP assay. by Dong Ma (584680)

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Representative TUNEL- and DAPI-stained sections from the abdominal aortas of young and old WT and smcKlf5^−/− mice following 28 days of Ang II infusion. by Dong Ma (584680)

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eIF5a affects the expression of mitochondrial dynamics–related genes. by Dong Ma (584680)

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Young (3 months) or old (18 months) WT and smcKlf5^−/− mice were infused with Ang II for 28 days. by Dong Ma (584680)

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Klf5 down-regulation leads to human VSMC senescence. by Dong Ma (584680)

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The spectral sensitivity of European seabass fed a 5% taurine diet. by Richard W. Brill (2337940)

PRMT5 activity aids in the sustained accumulation of lipid droplets during Mtb infection. by Salik Miskat Borbora (9381455)

Fbx15 phosphorylation decreases during oxidative stress treatment and shifts the cellular interaction site with SkpA to the nucleus. by Bastian Jöhnk (3127812)

Whole mount in situ hybridization (WISH) for <i>atp6v1e1</i> ohnologs in zebrafish embryos. by Lore Pottie (10994479)

“…<p>(A-C) Representative images demonstrating WISH for <i>atp6v1e1a</i> and <i>atp6v1e1b</i> at different developmental time points (24 hpf (n = 7), 48 hpf (n = 7), 72 hpf (n = 7), 120 hpf (n = 7) and 168 hpf (n = 7)). …”

Decreased Sho levels in ScN2a-Sho cells. by Joel C. Watts (195735)

“…(<b>C</b>) Quantification of Sho levels in ScN2a-Sho-1 (<i>n</i> = 10) and ScN2a-Sho-2 (<i>n</i> = 5) subclones revealed a significant decrease of 40–45% compared to the uninfected parental cell line (<i>n</i> = 15) (***<i>P</i><0.001).…”