Search alternatives:
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
nn decrease » _ decrease (Expand Search), gy decreased (Expand Search), b1 decreased (Expand Search)
we decrease » _ decrease (Expand Search), teer decrease (Expand Search), use decreased (Expand Search)
a decrease » _ decrease (Expand Search), _ decreased (Expand Search), _ decreases (Expand Search)
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8821
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8822
Attenuation of Quorum Sensing Regulated Virulence of <i>Pectobacterium carotovorum</i> subsp. <i>carotovorum</i> through an AHL Lactonase Produced by <i>Lysinibacillus</i> sp. Gs50
Published 2016“…Our results demonstrate the potential of <i>Lysinibacillus</i> sp. Gs50 as a preventive and curative biocontrol agent.…”
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8823
Quality of life and Beck Depression Inventory baseline and post CRT+MRT, CRT and RUHB.
Published 2024Subjects: -
8824
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8825
ΔQuality of life and ΔBeck Depression Inventory baseline and post CRT+MRT, CRT and RUHB.
Published 2024Subjects: -
8826
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8827
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8828
Role of the ACB domain and linker region in NMT2 activity.
Published 2020“…Human NMT2 enzyme was added at a concentration of 50nM in the presence of 5μM C<sub>14</sub>-CoA. …”
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8829
Pre-diabetes is associated with increased risk of heart failure among patients with atrial fibrillation
Published 2022“…</p> <p><strong>Results</strong>: Among 17,943 AF patients (mean age 75.5 years, 47% female), 3,711 (20.7%) had pre-diabetes and 10,127 (56.4%) had diabetes at baseline. …”
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8830
microRNA-34a-Mediated Down-Regulation of the Microglial-Enriched Triggering Receptor and Phagocytosis-Sensor TREM2 in Age-Related Macular Degeneration - Fig 6
Published 2016“…After vacuum evaporation of HFIP, aliquoted peptide was re-suspended with DMSO to 5 mM and diluted to 5 μM into the cell culture media; <b>(B)</b> cells were treated with a scrambled miRNA-34a sequence (miRNA-34a-<b>sc</b>; 30 nM;, control, <b>a-d</b>, middle row); or <b>(C)</b> with an LNA-stabilized miRNA-34a (30 nM; miRNA-34a stressed; <b>a-d</b>; bottom row); treatments were for 24 hr before incubation with 5 uM of Aβ42 (made up as in [<a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0150211#pone.0150211.ref033" target="_blank">33</a>]) for another 24 hr before assay; MG cells were subsequently stained using a murine amyloid beta MABN10 (red fluorescence λ<sub>max</sub>~650 nm; anti-Aβ antibody, clone W0-2; Millipore, Bellerica MA), a TREM-2 antibody (M-227): sc-48765 (green fluorescence; λ<sub>max</sub>~510 nm; Santa Cruz, Santa Cruz CA) or DAPI nuclear stain (blue fluorescence; λ<sub>max</sub>~470 nm) as in <b><a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0150211#pone.0150211.g003" target="_blank">Fig 3</a>;</b> arrows indicate Aβ42 uptake into MG cells; note decreased presence of TREM2 in miRNA-34a treated MG cells (bottom row, panel B) and decrease in ingested Aβ42 peptide within C8B4 cells (<b>C</b>; bottom row, panel <b>d</b>). …”
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8831
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8832
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8833
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8834
High-dose thiamine prevents brain lesions and prolongs survival of <i>Slc19a3</i>-deficient mice
Published 2017“…To investigate the molecular mechanisms of neurodegeneration in SLC19A3 deficiency and whether administration of high-dose thiamine prevents neurodegeneration, we generated homozygous Slc19a3 E314Q knock-in (KI) mice harboring the mutation corresponding to the human SLC19A3 E320Q, which is associated with the severe form of THMD2. …”
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8835
Normalized diatom species abundance for all species (A) and mean Fisher Information (B) for Foy Lake.
Published 2014“…At ∼4.5 ka species evenness decreases, and the system begins a ∼2 kyr gradual decrease in mean FI. …”
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8836
PRMT5 regulates EMT and invasion under hypoxia.
Published 2025“…<b>C)</b> Immunoblot showing decrease in histone marks HR38me2s and H4R3me2s upon vehicle (DMSO) vs. 5 µM GSK591 treatment in MCF7 cells under hypoxia. …”
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8837
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8838
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8839
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8840