Showing 20,301 - 20,320 results of 227,959 for search '(( 5 ((nn decrease) OR (mean decrease)) ) OR ( a ((point decrease) OR (a decrease)) ))', query time: 2.13s Refine Results
  1. 20301

    Dissolution proxies for holes 1207A, 1208A, and 1209A. by Adriane R. Lam (9127871)

    Published 2020
    “…<p>Dissolution intervals were approximated by calculating a ratio of susceptible to resistant species [<a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0234351#pone.0234351.ref077" target="_blank">77</a>]. …”
  2. 20302
  3. 20303

    Perturbation of the Charge Density between Two Bridged Mo<sub>2</sub> Centers: The Remote Substituent Effects by Tao Cheng (1235148)

    Published 2014
    “…X-ray structural analyses show that these complexes share the same molecular scaffold with the <i>para</i>-substituents (X) being about 8 Å away from the Mo<sub>2</sub> center. It is found that the remote substituents have the capability to tune the electronic properties of the complexes. …”
  4. 20304
  5. 20305

    Disparities in Hemoglobin A1c Testing During the Transition to Adulthood and Association With Diabetes Outcomes in Youth-Onset Type 1 and Type 2 Diabetes: The SEARCH for Diabetes i... by Katherine A Sauder (11126343)

    Published 2021
    “…For type 2 diabetes, not seeing an endocrinologist decreased odds of reporting ≥3 HbA1c tests/year over time (OR 0.19 [0.06-0.63]), but HbA1c testing frequency was not associated with HbA1c levels or microvascular complications.…”
  6. 20306

    Efficacy of Neonatal HBV Vaccination on Liver Cancer and Other Liver Diseases over 30-Year Follow-up of the Qidong Hepatitis B Intervention Study: A Cluster Randomized Controlled T... by Chunfeng Qu (114656)

    Published 2014
    “…The estimated efficacy of catch-up vaccination on HBsAg seroprevalence in early adulthood was 21% (95% CI 10%–30%), substantially weaker than that of the neonatal vaccination (72%, 95% CI 68%–75%). Receiving a booster at age 10–14 years decreased HBsAg seroprevalence if participants were born to HBsAg-positive mothers (hazard ratio [HR] = 0.68, 95% CI 0.47–0.97). …”
  7. 20307

    Effect of dilution and DMSO concentration on the interaction of NF-κB/DNA complex with the inhibitors. by Rodrigo Villagomez (685829)

    Published 2015
    “…<p><b>a)</b> Effect of 1) active compound <b>1</b>; 2) inactive compound <b>4</b>; B1) control complex NF-κB:DNA target and B2) free DNA target, at four different dilutions of the system: I) Initial conditions; II) Inhibitor at ¾, NF-κB at ¾ and DNA target to ½ of initial concentration; III) Diluting inhibitor to ½, NF-κB to ½ and DNA target to ¼ of initial concentration and IV) Diluting inhibitor at ½, NF-κB at ½ and DNA target at ¼ of initial concentration. …”
  8. 20308

    A model of a yeast cell with a simplified glycolysis pathway. by Albertas Janulevicius (732672)

    Published 2021
    “…<p>(A) A generalized core model of yeast glycolysis [<a href="http://www.ploscompbiol.org/article/info:doi/10.1371/journal.pcbi.1008547#pcbi.1008547.ref003" target="_blank">3</a>] considers the intracellular concentrations of the glycolytic intermediate fructose-1,6-bisphosphate (FBP), ATP and inorganic phosphate (P<sub>i</sub>), and four reactions (arrows): (i) a lumped upper glycolysis reaction that produces FBP from extracellular glucose with rate <i>v</i><sub>up</sub>, (ii) a lumped lower glycolysis reaction that generates ATP and the waste product ethanol (EtOH) at rate <i>v</i><sub>lo</sub>, (iii) ATPase reaction reflecting general ATP demand in the cell at rate <i>v</i><sub>atp</sub>, and (iv) reversible phosphate transport between the cytosol and the vacuole at rate <i>v</i><sub>p</sub>. …”
  9. 20309

    The Assembly of EDC4 and Dcp1a into Processing Bodies Is Critical for the Translational Regulation of IL-6 by Eri Seto (243456)

    Published 2015
    “…Interestingly, knocking down EDC4 or Dcp1a, which are components of P-bodies, severely reduced the production of IL-6, but not TNF-α in M1-THPs without decreasing the amount of IL-6 mRNA. …”
  10. 20310
  11. 20311

    MCPIP1 ribonuclease can bind and cleave <i>AURKA</i> mRNA in <i>MYCN</i>-amplified neuroblastoma cells by Iwona Nowak (9283821)

    Published 2021
    “…We showed that the levels of several transcripts involved in cell cycle progression decreased in BE(2)-C and KELLY cells overexpressing MCPIP1 in a ribonucleolytic activity-dependent manner. …”
  12. 20312
  13. 20313
  14. 20314
  15. 20315
  16. 20316
  17. 20317

    Cytochrome bd-II oxidase CyxA promotes the pathogenicity of <i>Klebsiella pneumoniae</i> by resisting oxidative stress by Xiao Xiao (99147)

    Published 2025
    “…Additionally, the expression levels of <i>cyxA</i> at 37°C and 41°C were significantly higher compared to 30°C, and the Δ<i>cyxA</i> strain exhibited significantly lower viable counts, elevated intracellular reactive oxygen species (ROS) levels, and decreased total antioxidant capacity (T-AOC) relative to WT at 37°C and 41°C. …”
  18. 20318

    Improving the Oxidative Stability of a High Redox Potential Fungal Peroxidase by Rational Design by Verónica Sáez-Jiménez (731925)

    Published 2015
    “…Substitution of these residues, located near the heme cofactor and the catalytic tryptophan, rendered a variant with a 7.8-fold decreased oxidative inactivation rate. …”
  19. 20319

    Image_3_T-cell infiltration in the central nervous system and their association with brain calcification in Slc20a2-deficient mice.TIF by Yi Zhang (9093)

    Published 2023
    “…<p>Primary familial brain calcification (PFBC) is a rare neurodegenerative and neuropsychiatric disorder characterized by bilateral symmetric intracranial calcification along the microvessels or inside neuronal cells in the basal ganglia, thalamus, and cerebellum. …”
  20. 20320

    Image_2_T-cell infiltration in the central nervous system and their association with brain calcification in Slc20a2-deficient mice.TIF by Yi Zhang (9093)

    Published 2023
    “…<p>Primary familial brain calcification (PFBC) is a rare neurodegenerative and neuropsychiatric disorder characterized by bilateral symmetric intracranial calcification along the microvessels or inside neuronal cells in the basal ganglia, thalamus, and cerebellum. …”