DataSheet_1_Transcriptional Profiling and Functional Analysis of N1/N2 Neutrophils Reveal an Immunomodulatory Effect of S100A9-Blockade on the Pro-Inflammatory N1 Subpopulation.doc... by Andreea C. Mihaila (11252988)

“…S100A9 inhibition with a specific small-molecule blocker, reduced CCL2, CCL3 and CCL5 chemokine expression and decreased MPO and MMP-9 activity, by interfering with the NF-kB signaling pathway. …”

Modafinil decreased EEG amplitude in the synchronized model. by Chang-Rui Chen (467755)

Regenerating axons are increased by ATRA and decreased by clodronate. by Valeria De La Rosa-Reyes (6658613)

“…(E) ATRA and clodronate liposome treatment, composite from 2 sections. Scale bar: 100 μm in A and B. (F) Bar and scatter plots of the number of axons per transverse section, sampled at approximately 200 μm distal to the cut site, showing mean ± SEM. …”

The Intracellular level of FLASH decreases during apoptosis. by Song Chen (69646)

As the number of infections <i>I</i> in a given county increases, the variance in exponential growth rate, Var(Δ<i>I</i>/<i>I</i>), decreases as . by Calvin Pozderac (10451496)

Increase of mitochondrial dysfunction and glycolysis by decreased NEFH. by Myoung Sook Kim (91321)

Matrix crosslinking decreases cell migration persistence. by Michael Pamonag (12295175)

Short-term depression decreases memory retention. by Alexander Seeholzer (6615989)

Recombinant 3xK-C100Flag monomer increased longer Aβ peptides. by Joo In Jung (650746)

“…Molecular mass (m/z) of monomeric 3xK-C100Flag for Aβ40 is 4588.43 (calculated m/z: 4589.27), for Aβ42 is 4773.07 (calculated m/z: 4773.51), and for Aβ45 is 5084.52 (calculated m/z: 5086.90). 3xK-C100Flag dimer and trimer showed decreased Aβ levels compared to the monomer. …”

MicroRNA-138 Regulates Hypoxia-Induced Endothelial Cell Dysfunction By Targeting S100A1 by Anagha Sen (482673)

“…Bioinformatic analysis suggested that microRNA -138 (MiR-138) could target the 3′UTR of S100A1. Patients with critical limb ischemia (CLI) or mice subjected to femoral artery resection (FAR) displayed increased MiR-138 levels and decreased S100A1 protein expression. …”

Purified recombinant WT-C100Flag dimer and trimer showed reduced Aβ production. by Joo In Jung (650746)

“…<p>(A) The purified recombinant WT-C100Flag monomer, dimer, and trimer eluates were loaded on a SDS-PAGE gel. …”

Sphere-forming efficiency decreases following radiation. by Vicky T. Nguyen (5029244)

GluN2A(D731N) reduces the agonist potency. by Kai Gao (120150)

“…(<b>C,E</b>) The composite glutamate (in the presence of 100 μM glycine) concentration-response curves reveal a significant decrease in glutamate potency in both di-heteromeric (<b>C</b>) and tri-heteromeric (<b>E</b>) GluN2A(D731N)-containing NMDARs compared to wild type receptors. …”

Intravenous treatment of naïve wild-type mice with PrP LSPCs decreased PrP^C mRNA and cell surface PrP^C protein at various time points. by Heather Bender (363996)

Data on 1pre-treatment1 and ¹post-treatment1 tracking1 tiles reflecting1decrease in rodent1 activity¹ as a result1 of 0.0051% brodifacoum^m mixed1 i... by Durr e Shahwar (17798007)

The substitution F447L is adaptive for J4_S52-HVR1, while it decreases infectivity of parental J4 and J4_ΔHVR1 HCVcc. by Christina Holmboe Olesen (11154333)

Table_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Image_4_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Table_1_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.docx by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”

Image_2_PE_PGRS31-S100A9 Interaction Promotes Mycobacterial Survival in Macrophages Through the Regulation of NF-κB-TNF-α Signaling and Arachidonic Acid Metabolism.TIF by Sheng Liu (279488)

“…Our results revealed that M. tb Rv1768 promotes mycobacterial survival in macrophages by regulating NF-κB-TNF-α signaling and arachidonic acid metabolism via S100A9. Disturbing the interaction between Rv1768 and S100A9 may be a potential therapeutic target for tuberculosis.…”