Showing 621 - 640 results of 27,215 for search '(( 5 ((nn decrease) OR (point decrease)) ) OR ( 100 ((teer decrease) OR (a decrease)) ))', query time: 0.76s Refine Results
  1. 621

    Knockdown of NRP1 increases while knockdown of NRP2 decreases formation of Schwann cell clusters. by Kasper C. D. Roet (444802)

    Published 2014
    “…SCs are shown in grey (S100 labeling). MCs are not labeled. Valid cell clusters were outlined with a blue line. …”
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    AICD transgenic mice show decreased cell proliferation in the SGZ of hippocampus. by Kaushik Ghosal (244754)

    Published 2010
    “…<p>Representative images of BrdU immunostaining in the subgranular zone (SGZ) of the dentate gyrus performed on 1.5-mont-old (<b>A</b>) 3-month-old (<b>C</b>) and 12-month old (<b>D</b>) wild-type and FeCγ25 animals one day after the final BrdU injection (100 mg/kg). …”
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    Table 1_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.xlsx by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
  17. 637

    Image 2_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.tif by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
  18. 638

    Image 1_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.tif by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
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