Table_1_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_8_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_9_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_4_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_7_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_13_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_10_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_6_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_2_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_3_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

Table_11_Trends of serum 25(OH) vitamin D and association with cardiovascular disease and all-cause mortality: from NHANES survey cycles 2001–2018.docx by Changlun Hu (17891735)

“…¹</p>Results<p>Survey data from 2001 to 2018 revealed a rise in serum 25(OH)D levels, from a weighted mean (95% CI) of 65.6 (63.8–67.4) nmol/L during 2001–2002 to 73.5 (70.4–76.5) nmol/L during 2017–2018, among US adults, while overall vitamin D deficiency rates remained stable (p = 0.152). …”

DataSheet_1_Biomarkers of postharvest resilience: unveiling the role of abscisic acid in table grapes during cold storage.docx by Ángela Navarro-Calderón (17065251)

“…‘Krissy’ were subjected to i) control (untreated); and ii) 1-MCP (1 µL L^-1; 12 hours; 15°C) and stored under two scenarios: i) 15 days at 0.5°C, followed by five days at 5.5°C to simulate shelf-life; and ii) 20 days at 5.5°C to simulate a higher storage temperature followed by shelf-life. …”

Gemcitabine and ATR inhibitor synergism and effects on the cell cycle and DNA damage in U-CHCF365 and UM-Chor1 (Batch 1) by Chordoma Foundation (4391989)

“…U-CHCF365 and UM-Chor1 cells were plated in a black walled, glass bottom, 96-well plate at 5,000 cells/well and 1,500 cells/well, respectively. …”

Data_Sheet_1_Soil Application of Almond Residue Biomass Following Black Soldier Fly Larvae Cultivation.docx by Robert Axelrod (6307505)

“…Under aerobic conditions, SPH addition increased the CO₂-accumulation by a factor of 5–6 compared to the non-amended soils in SL and CL, respectively. …”

Protocol: Temporary Pacemaker Verification and Troubleshooting by Jill Ley (4090672)

“…<p>(Figure 1)</p> <p>(Figure 2)</p> <p><strong>Determining the Stimulation Threshold</strong></p> <ul> <li>NOTE: a paced rhythm must be present and patient stable</li> <li>From 100% pacing, gradually decrease output for A or V, while watching monitor until you lose capture</li> <li>Now gradually increase output until 1:1 capture returns – this is the stimulation threshold</li> <li>This is the lowest amount of energy that will reliably capture the heart and pace</li> <li>Set mA 1.5-3 times above this threshold value for safety</li> <li>Repeat for other circuit (A or V)</li> <li>Note: the stimulation threshold should be checked DAILY by the MD/NP when pacing is in use; in the event 
the threshold value is rising, discuss with surgeon to determine if alternative pacing system is needed</li> </ul> <p><strong>Determining the Sensing Threshold</strong></p> <ul> <li>Note: the patient must have an underlying rhythm and tolerate a brief period without pacing to perform</li> <li>Set RATE at least 10 bpm below patient’s intrinsic heart rate (to allow sensing)</li> <li>Set OUTPUT at 0.1 mA (to avoid competitive pacing when device is made asynchronous briefly)</li> <li>Highlight SENSITIVITY (under Menu 1)</li> <li>Decrease SENSITIVITY: Slowly turn dial counter-clockwise until pace indicator flashes regularly (asynchronously)</li> <li>Increase SENSITIVITY: Slowly turn dial clockwise until sense indicator flashes again (when sensing resumes, this is the sensing threshold – the smallest complex that is reliably recognized, thus inhibiting the device)</li> <li>Set SENSITIVITY < half this threshold value for safety</li> <li>Repeat for other circuit (A or V)</li> <li>Restore previous rate and output values</li> </ul> <p><strong>Rapid Atrial (Overdrive) Pacing – PERFORMED BY MD/NP/CNS ONLY</strong></p> <ul> <li>Used to “overdrive pace” and terminate some atrial reentrant tachyarrhythmias, especially atrial flutter</li> <li>ALWAYS VERIFY correct wire connections to avoid stimulating the ventricles at a rapid rate!…”

Susceptibility to HIV-1 infection in primary macrophages depends on differentiation stimuli. by Roger Badia (482283)

“…<p><b>(A)</b> Evaluation of cell proliferation by Ki67 staining. …”

Functional study of moDC maturation. by Laurent Gorvel (279197)

“…<b>B</b>, moDCs stimulated with <i>T. whipplei</i> (a), <i>B. abortus</i> (b), <i>C. burnetii</i> (c), <i>O. tsutsugamushi</i> (d) or <i>E. coli</i> LPS (e) were co-cultured with CFSE-labeled autologous T-lymphocytes for 5 days. …”

Thapsigargin-induced differentiation is associated with increased KGFR expression. by Francesca Belleudi (233982)

“…<p>(A) HaCaT cells were treated with TG 0.5 µM for 1 h at 37°C following by incubation at 37°C for 48 h. …”

Administration of CORM-2 does not improve m-RCT in vivo in diabetic mice. by Karen Alejandra Méndez-Lara (5819492)

“…In all panels, results are the mean ± SEM of 5 mice and are expressed in % vs. injected dose. …”

Evaluation of cesarean delivery rates in different levels of hospitals in Jiangsu Province, China, using the 10-Group classification system by Ning Gu (226838)

“…In non-referral hospitals, CD rate in Group 5 and the proportion of Group 5 to the overall population were also significantly higher than those in referral hospitals (98.5% vs. 92.5%, <i>p</i> < .001; and 21.0% vs. 14.5%, <i>p</i> < .001).…”