Flame-Retardant and Self-Healing Waterborne Polyurethane Based on Organic Selenium by Xiang Shang (13257384)

“…The limiting oxygen index of SWPU-4 was improved to 28.5% with excellent UL-94 level (V-0) and self-healing efficiency (91.25%, after being healed in the photoreactor for 30 min at room temperature), together with high mechanical properties (tensile strength was 18.5 MPa and elongation at break was 869.63%), and the total heat release (THR) for SWPU-4 (49.28 MJ/m²) could decrease to 23.80% of the THR for the original waterborne polyurethane WPU (64.67 MJ/m²). …”

Impact of PFOS Exposure on Murine Fetal Hematopoietic Stem Cells, Associated with Intrauterine Metabolic Perturbation by Wang Ka LEE (20876477)

“…Flow cytometric analysis confirmed increased HSC populations and progenitor activation for megakaryocyte, erythrocyte, and lymphocyte lineages. The CFU assay showed a significant increase in BFU-E and CFU-G, but a decrease in CFU-GM in FL-HSCs from the H-PFOS group, indicating altered differentiation potential. …”

Flame-Retardant and Self-Healing Waterborne Polyurethane Based on Organic Selenium by Xiang Shang (13257384)

“…The limiting oxygen index of SWPU-4 was improved to 28.5% with excellent UL-94 level (V-0) and self-healing efficiency (91.25%, after being healed in the photoreactor for 30 min at room temperature), together with high mechanical properties (tensile strength was 18.5 MPa and elongation at break was 869.63%), and the total heat release (THR) for SWPU-4 (49.28 MJ/m²) could decrease to 23.80% of the THR for the original waterborne polyurethane WPU (64.67 MJ/m²). …”

Flame-Retardant and Self-Healing Waterborne Polyurethane Based on Organic Selenium by Xiang Shang (13257384)

“…The limiting oxygen index of SWPU-4 was improved to 28.5% with excellent UL-94 level (V-0) and self-healing efficiency (91.25%, after being healed in the photoreactor for 30 min at room temperature), together with high mechanical properties (tensile strength was 18.5 MPa and elongation at break was 869.63%), and the total heat release (THR) for SWPU-4 (49.28 MJ/m²) could decrease to 23.80% of the THR for the original waterborne polyurethane WPU (64.67 MJ/m²). …”

Compliance of Health Care Workers with Hand Hygiene Practices: Independent Advantages of Overt and Covert Observers by Sung-Ching Pan (103241)

“…Covert observation offers the opportunity to decrease observer bias. In this study we conducted a one year hospital-wide HH promotion program that included medical students (MS) as covert observers.…”

Fluorescent Nitric Oxide Detection by Copper Complexes Bearing Anthracenyl and Dansyl Fluorophore Ligands by Mi Hee Lim (1474606)

“…A decrease in fluorescence compared to that of the free ligands (<b>1</b>−<b>5</b>) was measured following the formation of the corresponding five Cu^II complexes <b>6</b>−<b>10</b>. …”

Figure S1 - Immune Responses and Hypercoagulation in ERT for Pompe Disease Are Mutation and rhGAA Dose Dependent by Sushrusha Nayak (371224)

“…<p><b>A</b>) Anti-rhGAA IgG2b in 1 mg/kg rhGAA injected GAA-/- 129SVE mice <b>B</b>) Anti-rhGAA IgG2b in 5 mg/kg rhGAA injected GAA-/- 129SVE mice <b>C</b>) Anti-rhGAA IgG2b in 20 mg/kg rhGAA injected GAA-/- 129SVE mice <b>D</b>) Anti-rhGAA IgG2b in 20 mg/kg rhGAA injected P545L mice <b>E</b>) Anti-rhGAA IgM in 1 mg/kg rhGAA injected GAA-/- 129SVE mice tested weekly <b>F</b>) Anti-rhGAA IgM in 5 mg/kg rhGAA injected GAA-/- 129SVE mice <b>G</b>) Anti-rhGAA IgM in 20 mg/kg rhGAA injected GAA-/- 129SVE mice <b>H</b>) Anti-rhGAA IgM response in 20 mg/kg rhGAA injected P545L mice. …”

Temporal Trends in Incidence of Myocardial Infarction and Ischemic Stroke by Socioeconomic Position in Sweden 1987–2010 by Ninoa Malki (485761)

“…</p><p>Conclusion</p><p>There was a decrease in myocardial infarction and ischemic stroke incidence over time among men but no significant change for women. …”

Fluorescent Nitric Oxide Detection by Copper Complexes Bearing Anthracenyl and Dansyl Fluorophore Ligands by Mi Hee Lim (1474606)

“…A decrease in fluorescence compared to that of the free ligands (<b>1</b>−<b>5</b>) was measured following the formation of the corresponding five Cu^II complexes <b>6</b>−<b>10</b>. …”

Exploring Beyond Numeric Weight Loss: The Metabolic Effects of Semaglutide. by Sara, Sokary

“…Also, it enhanced glycemic control, as evidenced by significant reductions in hemoglobin A1C (HbA1c) with the 0.5 mg and 1.0 mg doses. From a baseline range of 8.1–8.7%, 0.5mg dose lowered HbA1c by 1.2–1.5%, while the 1.0 mg dose reduced it by 1.4–1.8%. …”
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Synthesis and characterization of stir casted Cu-ZrO2-Graphite hybrid metal matrix composites for thermal management structures by Mittal, Prateek

“…ZrO2 and Graphite reinforcements were added (0.5–0.5, 1.0–1.0, 1.5–1.5 and 2.0–2.0 wt %) in the four composite samples prepared via stir casting. …”
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Hormone-sensitive BCa cells (MCF-7) comprise 3 subpopulations, distinguished by ER-α46 expression levels. by John J. Kim (11191609)

“…<b>C</b> Scatter plots of ER-α66 and ER-α46 expression levels in individual MCF-7 cells (*p = 0.03, ***p < 0.0001, n_{ER-α66_only} = 329 cells, n_both = 26 cells). Red lines represent mean values. <b>D</b> Top: scatter plots indicate that mean ER-α66 expression level is decreased in ER-α66⁺ subpopulation by TAM (***p < 0.0001, n_neg = 604, n_TAM = 227). …”

Image_3_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_2_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_4_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_1_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_3_RETRACTED: Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_2_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_2_RETRACTED: Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.jpeg by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Image_1_Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”