Showing 11,901 - 11,920 results of 102,308 for search '(( 5 ((teer decrease) OR (a decrease)) ) OR ( 50 ((mean decrease) OR (nn decrease)) ))', query time: 1.42s Refine Results
  1. 11901

    Image_2_Allopregnanolone Modulates GABAAR-Dependent CaMKIIδ3 and BDNF to Protect SH-SY5Y Cells Against 6-OHDA-Induced Damage.JPEG by Tongtong Wang (775312)

    Published 2022
    “…As a result, APα-induced an increase in the number of TH-positive SH-SY5Y cells might be mediated through GABAAR via Ca<sup>2+</sup>/CaM/CaMKIIδ3/BDNF (CDK1) signaling pathway, which would ultimately facilitate to elucidate PD pathogenesis and hold a promise as an alternative therapeutic target for PD.…”
  2. 11902

    Neuromuscular junction activity differs in overexpression <i>versus</i> single-copy/knock-in models. by Saba N. Baskoylu (5828636)

    Published 2018
    “…While <i>sod-1G85R</i><sup><i>M</i></sup> and <i>sod-1A4V</i><sup><i>M</i></sup> models appear slightly hypersensitive, this difference was not significant. …”
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    Miz1 Deficiency in the Mammary Gland Causes a Lactation Defect by Attenuated Stat5 Expression and Phosphorylation by Adrián Sanz-Moreno (172726)

    Published 2014
    “…Further analysis revealed decreased Stat5 activity in <i>Miz1</i>Δ<i>POZ</i> mammary glands and an attenuated expression of Stat5 targets. …”
  13. 11913

    Data_Sheet_1_A Dual Role for Death Receptor 5 in Regulating Cardiac Fibroblast Function.PDF by Miles A. Tanner (11353644)

    Published 2021
    “…To investigate the impact of DR5 regulation of fibroblasts in vivo, a chronic isoproterenol administration model of heart failure was used. …”
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    <em>GCN5</em> Is a Positive Regulator of Origins of DNA Replication in <em>Saccharomyces cerevisiae</em> by Maria Claudia Espinosa (253850)

    Published 2010
    “…Here we show that the deletion of <em>GCN5</em> decreases the stability of mini-chromosomes; that the tethering of <em>Gcn5p</em> to a crippled origin of replication stimulates its activity; that high dosage of <em>GCN5</em> suppresses conditional phenotypes caused by mutant alleles of <em>bona fide</em> replication factors, <em>orc2-1</em>, <em>orc5-1</em> and <em>mcm5-461</em>. …”