Table_5_Identification of Distinct Unmutated Chronic Lymphocytic Leukemia Subsets in Mice Based on Their T Cell Dependency.docx by Simar Pal Singh (5744543)

“…Conversely, in vivo T cell-dependent immunization decreased the proportions of V_H11/Vκ14-expressing CLL. …”

The Design, Synthesis, and Characterizations of Spore Germination Inhibitors Effective against an Epidemic Strain of Clostridium difficile by Shiv K. Sharma (1945762)

“…To circumvent this problem, a series of cholic acid amides were synthesized and tested against R20291. …”

Simulation results for different proportions of normal stem cells (generate cells with normal inhibitor levels) to abnormal stem cells (generate cells with no inhibitor). by Claire Miller (3830644)

Data_Sheet_5_D-galactose Intake Alleviates Atopic Dermatitis in Mice by Modulating Intestinal Microbiota.DOCX by Dong-Yeon Kim (770670)

Image_1_CD44v9 Induces Stem Cell-Like Phenotypes in Human Cholangiocarcinoma.TIF by Nattawan Suwannakul (8924774)

“…Therefore, in this study, we examined the potential roles of CD44v9 in CCA cells to understand the carcinogenic mechanism.…”

Image_2_CD44v9 Induces Stem Cell-Like Phenotypes in Human Cholangiocarcinoma.TIF by Nattawan Suwannakul (8924774)

“…Therefore, in this study, we examined the potential roles of CD44v9 in CCA cells to understand the carcinogenic mechanism.…”

The growth of co-transformed cells in the peritoneal cavity alters their cellular gene expression to mirror that of PEL biopsies. by Mitchell Hayes (5334887)

Table_5_The metabolome of human milk is altered differentially by Holder pasteurization and high hydrostatic pressure processing.docx by Léa Chantal Tran (12471495)

“…Decreases were more strongly noted in HP samples rather than in HoP ones. …”

Image_5_RETRACTED: Oxidized LDL Causes Endothelial Apoptosis by Inhibiting Mitochondrial Fusion and Mitochondria Autophagy.JPEG by Jia Zheng (140297)

“…Opa1 overexpression reversed this effect by increasing endothelial cell viability and decreasing apoptosis. Interestingly, inhibition of mitophagy or mitochondrial fusion through transfection of siRNAs against Atg5 or Mfn2, respectively, abolished the protective effects of Opa1. …”

Video_5_C1q Regulates Horizontal Cell Neurite Confinement in the Outer Retina.MP4 by Courtney A. Burger (9527840)

Data_Sheet_5_Microbial community structure dynamics of invasive bullfrog with meningitis-like infectious disease.pdf by Wengang Li (300649)

“…It is highly contagious and has a high mortality rate. In this study, we sampled and sequenced microbiomes from oral and intestinal samples of five normal and five diseased bullfrogs. …”

DataSheet5_LMO3 downregulation in PCa: A prospective biomarker associated with immune infiltration.PDF by Wenchao Xu (2629939)

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

Supplementary Material for: Somatic variants and exon-level copy number changes in five hyperplastic-stage oral leukoplakias. by Anne A. (13992672)

“…Oral leukoplakia (OL), an oral potentially malignant disorder begins with a hyperplastic/hyperkeratotic stage at which no genome-scale somatic single nucleotide variant profiles have been described so far. We performed exome sequencing of five cases at this stage with no evidence of dysplasia to identify genetic alterations (exon-level copy number alterations, indels, and single nucleotide variants), their association with transcript levels and relationship with oral cancer susceptibility. …”

The effects of losartan on cytomegalovirus infection in human trabecular meshwork cells by Jin A. Choi (559035)

“…</p><p>Results</p><p>CMV infection significantly increased the concentrations of the secreted TGF-β1 at 3, 5, and 7 day post infection in TM cells. Treatment with dexamethasone or losartan significantly decreased the levels of TGF-β1, whereas treatment with ganciclovir did not affect TGF-β1 levels. …”