Showing 641 - 660 results of 23,476 for search '(( 5 ((wt decrease) OR (nn decrease)) ) OR ( 100 ((teer decrease) OR (a decrease)) ))', query time: 0.91s Refine Results
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    ApoA-I inhibits leukocyte adhesion to endothelial cells <i>in vivo</i>. by Scott J. Cameron (838315)

    Published 2015
    “…(B) ApoA-I knockout mice (apoA-I <sup>-/-</sup>) have low plasma HDL concentration compared to WT mice and show increased adhesion of leukocytes to the vascular endothelium <i>in vivo</i> (decreased rolling velocity) compared to WT mice or apoA-I <sup>-/-</sup> mice injected with apo-AI (0.5mg, intraperitoneal) 24 hours before. …”
  3. 643

    eNOS phosphorylation increased intracellular ROS generation and decreased cell viability in BH4-depleted cells. by Hu Peng (396074)

    Published 2015
    “…(B) VEGF-mediated eNOS phosphorylation resulted a significant ROS increase in eNOS overexpressing HEK 293 cells (n = 5 fields, *, <i>p</i> < 0.05). (C) eNOS phosphorylation decreased cellular viability in BH4-depleted cells. …”
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    Cellular stress decreases TDP-43 levels in patient neurons with the TDP-43 A90V mutation. by Zhijun Zhang (315079)

    Published 2013
    “…The expression of transfected TDP-43 WT or TDP-43 A90V is induced by tetracycline. The A90V mutation alone is insufficient to decrease TDP-43 level in this assay. …”
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    Delphinidin alters VEGF-A splicing to increase VEGF-A<sub>165</sub>b and decrease total VEGF-A expression. by Megan Stevens (3964886)

    Published 2019
    “…<p><b>A)</b> Treatment of podocytes with delphinidin chloride (10 μg/ml) under normal glucose (NG; 5 mM glucose + 25 mM mannitol) and high glucose (HG; 30 mM glucose, 1 ng/ml TNFα, 1 ng/ml IL-6, and 100 nM insulin) for 48 hrs increased the protein expression of VEGF-A<sub>165</sub>b relative to total VEGF-A<sub>165</sub> (quantified in <b>B</b>; *p<0.05 vs NG, †p<0.05 vs HG; n = 3 biological repeats; One-way ANOVA with Bonferroni post-hoc test for comparison between pairs; <b>A</b>—the same blot was first probed with VEGF-A<sub>165</sub>b before stripping and reprobing with panVEGF-A). …”
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    Table 1_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.xlsx by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
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    Image 2_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.tif by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
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    Image 1_Atractylenolide-I restore intestinal barrier function by targeting the S100A9/AMPK/mTOR signaling pathway.tif by Chen Chen (6544)

    Published 2025
    “…Mechanistically, recombinant human S100A9 protein was found to induce a decrease in intracellular Ca<sup>2+</sup> concentration, while AT-1 regulated the expression of tight junction proteins via modulation of the AMPK/mTOR signaling pathway. …”
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